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How to reduce malpractice risk with better documentation
Typicals vs atypicals
“Corticosteroid psychosis: Stop therapy or add psychotropics?” (Med/Psych Update, Current Psychiatry, January 2010) provided excellent information, especially for consultation-liaison psychiatrists. I understand studies have been funded to show efficacy for atypicals, but can the authors point to studies that show efficacy for perphenazine, haloperidol, or other typical antipsychotics? My guess is that typicals and atypicals essentially are equivalent in corticosteroid-induced acute mania.
Corey Yilmaz, MD
Adult and child psychiatrist
Buckeye, AZ
The authors respond
Dr. Yilmaz is correct that typical antipsychotics would be useful for off-label treatment of corticosteroid-induced psychosis. Three publications provide further detail about using these medications for corticosteroid-induced psychosis. A case series by Wada et al1 and a case report by Ahmad and Rasul2 report on the efficacy of low-dose (1 mg/d to 4 mg/d) haloperidol in treating patients who developed acute psychosis following initiation of a corticosteroid. These patients showed a rapid and marked improvement with haloperidol. A brief report by Bloch et al3 describes a patient prophylactically treated with chlorpromazine, 150 mg, before receiving high-dose methylprednisone, initiated at 1 g and titrated over 10 days. The patient did not exhibit any psychiatric symptoms but did develop hypomania when chlorpromazine was stopped, which resolved with readministration of this medication. We did not find any case reports on perphenazine.
Andrew J. Muzyk, PharmD
Assistant professor
Campbell University School of Pharmacy
Clinical specialist in internal medicine/psychiatry
Department of pharmacy, Duke University Hospital
Shannon Holt, PharmD
Clinical pharmacist
Jane P. Gagliardi, MD
Assistant professor of psychiatry and behavioral sciences
Assistant professor of medicine
Duke University School of Medicine
Durham, NC
1. Wada K, Yamada N, Suzuki H, et al. Recurrent cases of corticosteroid-induced mood disorder: clinical characteristics and treatment. J Clin Psychiatry. 2000;61(4):261-267.
2. Ahmad M, Rasul FM. Steroid-induced psychosis treated with haloperidol in a patient with active chronic obstructive pulmonary disorder. Am J Emerg Med. 1999;17:735.-
3. Bloch M, Gur E, Shalev A. Chlorpromazine prophylaxis of steroid-induced psychosis. Gen Hosp Psychiatry. 1994;16:42-44.
“Corticosteroid psychosis: Stop therapy or add psychotropics?” (Med/Psych Update, Current Psychiatry, January 2010) provided excellent information, especially for consultation-liaison psychiatrists. I understand studies have been funded to show efficacy for atypicals, but can the authors point to studies that show efficacy for perphenazine, haloperidol, or other typical antipsychotics? My guess is that typicals and atypicals essentially are equivalent in corticosteroid-induced acute mania.
Corey Yilmaz, MD
Adult and child psychiatrist
Buckeye, AZ
The authors respond
Dr. Yilmaz is correct that typical antipsychotics would be useful for off-label treatment of corticosteroid-induced psychosis. Three publications provide further detail about using these medications for corticosteroid-induced psychosis. A case series by Wada et al1 and a case report by Ahmad and Rasul2 report on the efficacy of low-dose (1 mg/d to 4 mg/d) haloperidol in treating patients who developed acute psychosis following initiation of a corticosteroid. These patients showed a rapid and marked improvement with haloperidol. A brief report by Bloch et al3 describes a patient prophylactically treated with chlorpromazine, 150 mg, before receiving high-dose methylprednisone, initiated at 1 g and titrated over 10 days. The patient did not exhibit any psychiatric symptoms but did develop hypomania when chlorpromazine was stopped, which resolved with readministration of this medication. We did not find any case reports on perphenazine.
Andrew J. Muzyk, PharmD
Assistant professor
Campbell University School of Pharmacy
Clinical specialist in internal medicine/psychiatry
Department of pharmacy, Duke University Hospital
Shannon Holt, PharmD
Clinical pharmacist
Jane P. Gagliardi, MD
Assistant professor of psychiatry and behavioral sciences
Assistant professor of medicine
Duke University School of Medicine
Durham, NC
“Corticosteroid psychosis: Stop therapy or add psychotropics?” (Med/Psych Update, Current Psychiatry, January 2010) provided excellent information, especially for consultation-liaison psychiatrists. I understand studies have been funded to show efficacy for atypicals, but can the authors point to studies that show efficacy for perphenazine, haloperidol, or other typical antipsychotics? My guess is that typicals and atypicals essentially are equivalent in corticosteroid-induced acute mania.
Corey Yilmaz, MD
Adult and child psychiatrist
Buckeye, AZ
The authors respond
Dr. Yilmaz is correct that typical antipsychotics would be useful for off-label treatment of corticosteroid-induced psychosis. Three publications provide further detail about using these medications for corticosteroid-induced psychosis. A case series by Wada et al1 and a case report by Ahmad and Rasul2 report on the efficacy of low-dose (1 mg/d to 4 mg/d) haloperidol in treating patients who developed acute psychosis following initiation of a corticosteroid. These patients showed a rapid and marked improvement with haloperidol. A brief report by Bloch et al3 describes a patient prophylactically treated with chlorpromazine, 150 mg, before receiving high-dose methylprednisone, initiated at 1 g and titrated over 10 days. The patient did not exhibit any psychiatric symptoms but did develop hypomania when chlorpromazine was stopped, which resolved with readministration of this medication. We did not find any case reports on perphenazine.
Andrew J. Muzyk, PharmD
Assistant professor
Campbell University School of Pharmacy
Clinical specialist in internal medicine/psychiatry
Department of pharmacy, Duke University Hospital
Shannon Holt, PharmD
Clinical pharmacist
Jane P. Gagliardi, MD
Assistant professor of psychiatry and behavioral sciences
Assistant professor of medicine
Duke University School of Medicine
Durham, NC
1. Wada K, Yamada N, Suzuki H, et al. Recurrent cases of corticosteroid-induced mood disorder: clinical characteristics and treatment. J Clin Psychiatry. 2000;61(4):261-267.
2. Ahmad M, Rasul FM. Steroid-induced psychosis treated with haloperidol in a patient with active chronic obstructive pulmonary disorder. Am J Emerg Med. 1999;17:735.-
3. Bloch M, Gur E, Shalev A. Chlorpromazine prophylaxis of steroid-induced psychosis. Gen Hosp Psychiatry. 1994;16:42-44.
1. Wada K, Yamada N, Suzuki H, et al. Recurrent cases of corticosteroid-induced mood disorder: clinical characteristics and treatment. J Clin Psychiatry. 2000;61(4):261-267.
2. Ahmad M, Rasul FM. Steroid-induced psychosis treated with haloperidol in a patient with active chronic obstructive pulmonary disorder. Am J Emerg Med. 1999;17:735.-
3. Bloch M, Gur E, Shalev A. Chlorpromazine prophylaxis of steroid-induced psychosis. Gen Hosp Psychiatry. 1994;16:42-44.
Communicating with deaf patients: 10 tips to deliver appropriate care
When treating deaf psychiatric patients, appropriate psychiatric care is possible when you maintain an awareness of deaf culture and language. Consider these 10 points:
1. Certified American Sign Language (ASL) interpreters are necessary. The Americans with Disabilities Act requires that all health care providers offer “auxiliary aids and services to provide effective communication.” For deaf patients, often this means an interpreter. Certified interpreters:
- have passed fluency examinations
- are culturally competent
- follow standards of practice
- have a code of ethics
- are required to pursue continuing education to keep their skills sharp and up to date.
Visit the Registry of Interpreters for the Deaf at www.rid.org to search by state for freelance qualified ASL interpreters and interpreting agencies. Be aware that clinicians must pay for the interpreting service, which is not reimbursed by insurance. Rates vary by region.
2. ASL fluency levels vary. Approximately 90% of deaf children are born to hearing parents, and exposure to ASL may be delayed or minimal. As a result, the presence of a fluent ASL interpreter does not guarantee patient comprehension. The patient may not understand the questions being asked resulting in incorrect endorsement or denial of symptoms. Dysfluent patients’ language may mimic that of a patient with a thought disorder or intellectual disability, resulting in misdiagnosis.1
3. Meet the interpreter before the session. Discuss your goals and explain the meaning of psychiatric terms and symptoms to help him or her communicate your message and interpret the patient’s response.2
ASL is not visual English. Some concepts do not translate into ASL and need to be modified or omitted by interpreters. English idioms often used in diagnostic interviews, such as “feeling blue” or “feeling keyed up or on edge,” do not have exact ASL translations.
An interpreter has to use his or her judgment on how to translate these concepts in ASL. Some symptoms such as “panic attack” and “auditory hallucination” do not have corresponding signs in ASL.
Interpreters may convey these concepts by describing or even acting them out. Ambiguity is difficult to maintain in ASL. Interpreters often have to give examples or lists of possible choices to communicate a concept. Some open-ended questions must be transformed into multiple-choice questions, which can be leading or narrow potential responses.
4. Be aware of privacy. Certified interpreters are required to maintain confidentiality. However, a deaf patient might not have the same interpreter over the course of treatment. The deaf community is close-knit, and a patient using multiple interpreters may worry about confidentiality. Deaf people are likely to use interpreters in a variety of settings and may feel uncomfortable working with the same interpreter in a psychiatric setting and other social situations. The deaf patient always has final say over whether or not to work with a particular interpreter.
5. Avoid using families as interpreters. Even if they are fluent in ASL, family members can distort examination findings and will eliminate confidentiality. If a family member insists on serving as an interpreter, discuss his or her reasons. For example, some families use a form of idiosyncratic gestures often called “home signs” instead of ASL. In this case, you would need a family member present because an ASL interpreter would not be familiar with these. In other situations, you may have to educate family members about why a certified interpreter is more appropriate.
6. Don’t rely on written English. A typical deaf person of normal intelligence has a fourth-grade reading level. Self-report measures of symptoms often are written for an eighth-grade reading or higher level. In psychiatry, subtle nuances in communication are critical, and relying on written English could cause misunderstandings.
Do not use written notes passed back and forth with your patient to avoid the expense of an ASL interpreter. Physicians have been successfully sued for refusing to hire interpreters in the hope of “getting by” with written notes. In these cases the judgments against the physicians were not covered by malpractice insurance.
7. Don’t overpathologize. ASL is an expressive and dramatic language. In addition, deaf persons may have different personal space boundaries than hearing persons. Don’t mistake these cultural norms as evidence of a mood disorder or character pathology.
8. Be cautious when assessing a deaf person for psychosis and “hearing-based” phenomena such as auditory hallucinations. Psychotic disorders historically have been overdiagnosed in deaf patients when clinicians rely solely on subjective reports of symptoms. Instead, err on the side of caution unless you identify objective, observable evidence (such as bizarre behavior or clearly stated delusional beliefs).1
9. Clarify. Deaf persons—just like hearing persons—do not like to appear unknowledgeable. A patient may “nod along,” leading you to inaccurate conclusions and misdiagnosis. Use open-ended questions to elicit a full description of symptoms.
10. Be patient and plan ahead. Schedule longer sessions—as much as twice as long—to allow extra time for interpretation and double-checking comprehension. Remember that each question has to be asked, interpreted, answered, and interpreted again. Follow-up questions may be necessary to ensure comprehension.
1. Glickman N. Cognitive-behavioral therapy for deaf and hearing persons with language and learning challenges. New York, NY: Routledge; 2009.
2. Leigh IW, Corbett CA, Gutman V, et al. Providing psychological services to deaf individuals: a response to new perceptions of diversity. Prof Psychol Res Pr. 1996;4:364-371.
When treating deaf psychiatric patients, appropriate psychiatric care is possible when you maintain an awareness of deaf culture and language. Consider these 10 points:
1. Certified American Sign Language (ASL) interpreters are necessary. The Americans with Disabilities Act requires that all health care providers offer “auxiliary aids and services to provide effective communication.” For deaf patients, often this means an interpreter. Certified interpreters:
- have passed fluency examinations
- are culturally competent
- follow standards of practice
- have a code of ethics
- are required to pursue continuing education to keep their skills sharp and up to date.
Visit the Registry of Interpreters for the Deaf at www.rid.org to search by state for freelance qualified ASL interpreters and interpreting agencies. Be aware that clinicians must pay for the interpreting service, which is not reimbursed by insurance. Rates vary by region.
2. ASL fluency levels vary. Approximately 90% of deaf children are born to hearing parents, and exposure to ASL may be delayed or minimal. As a result, the presence of a fluent ASL interpreter does not guarantee patient comprehension. The patient may not understand the questions being asked resulting in incorrect endorsement or denial of symptoms. Dysfluent patients’ language may mimic that of a patient with a thought disorder or intellectual disability, resulting in misdiagnosis.1
3. Meet the interpreter before the session. Discuss your goals and explain the meaning of psychiatric terms and symptoms to help him or her communicate your message and interpret the patient’s response.2
ASL is not visual English. Some concepts do not translate into ASL and need to be modified or omitted by interpreters. English idioms often used in diagnostic interviews, such as “feeling blue” or “feeling keyed up or on edge,” do not have exact ASL translations.
An interpreter has to use his or her judgment on how to translate these concepts in ASL. Some symptoms such as “panic attack” and “auditory hallucination” do not have corresponding signs in ASL.
Interpreters may convey these concepts by describing or even acting them out. Ambiguity is difficult to maintain in ASL. Interpreters often have to give examples or lists of possible choices to communicate a concept. Some open-ended questions must be transformed into multiple-choice questions, which can be leading or narrow potential responses.
4. Be aware of privacy. Certified interpreters are required to maintain confidentiality. However, a deaf patient might not have the same interpreter over the course of treatment. The deaf community is close-knit, and a patient using multiple interpreters may worry about confidentiality. Deaf people are likely to use interpreters in a variety of settings and may feel uncomfortable working with the same interpreter in a psychiatric setting and other social situations. The deaf patient always has final say over whether or not to work with a particular interpreter.
5. Avoid using families as interpreters. Even if they are fluent in ASL, family members can distort examination findings and will eliminate confidentiality. If a family member insists on serving as an interpreter, discuss his or her reasons. For example, some families use a form of idiosyncratic gestures often called “home signs” instead of ASL. In this case, you would need a family member present because an ASL interpreter would not be familiar with these. In other situations, you may have to educate family members about why a certified interpreter is more appropriate.
6. Don’t rely on written English. A typical deaf person of normal intelligence has a fourth-grade reading level. Self-report measures of symptoms often are written for an eighth-grade reading or higher level. In psychiatry, subtle nuances in communication are critical, and relying on written English could cause misunderstandings.
Do not use written notes passed back and forth with your patient to avoid the expense of an ASL interpreter. Physicians have been successfully sued for refusing to hire interpreters in the hope of “getting by” with written notes. In these cases the judgments against the physicians were not covered by malpractice insurance.
7. Don’t overpathologize. ASL is an expressive and dramatic language. In addition, deaf persons may have different personal space boundaries than hearing persons. Don’t mistake these cultural norms as evidence of a mood disorder or character pathology.
8. Be cautious when assessing a deaf person for psychosis and “hearing-based” phenomena such as auditory hallucinations. Psychotic disorders historically have been overdiagnosed in deaf patients when clinicians rely solely on subjective reports of symptoms. Instead, err on the side of caution unless you identify objective, observable evidence (such as bizarre behavior or clearly stated delusional beliefs).1
9. Clarify. Deaf persons—just like hearing persons—do not like to appear unknowledgeable. A patient may “nod along,” leading you to inaccurate conclusions and misdiagnosis. Use open-ended questions to elicit a full description of symptoms.
10. Be patient and plan ahead. Schedule longer sessions—as much as twice as long—to allow extra time for interpretation and double-checking comprehension. Remember that each question has to be asked, interpreted, answered, and interpreted again. Follow-up questions may be necessary to ensure comprehension.
When treating deaf psychiatric patients, appropriate psychiatric care is possible when you maintain an awareness of deaf culture and language. Consider these 10 points:
1. Certified American Sign Language (ASL) interpreters are necessary. The Americans with Disabilities Act requires that all health care providers offer “auxiliary aids and services to provide effective communication.” For deaf patients, often this means an interpreter. Certified interpreters:
- have passed fluency examinations
- are culturally competent
- follow standards of practice
- have a code of ethics
- are required to pursue continuing education to keep their skills sharp and up to date.
Visit the Registry of Interpreters for the Deaf at www.rid.org to search by state for freelance qualified ASL interpreters and interpreting agencies. Be aware that clinicians must pay for the interpreting service, which is not reimbursed by insurance. Rates vary by region.
2. ASL fluency levels vary. Approximately 90% of deaf children are born to hearing parents, and exposure to ASL may be delayed or minimal. As a result, the presence of a fluent ASL interpreter does not guarantee patient comprehension. The patient may not understand the questions being asked resulting in incorrect endorsement or denial of symptoms. Dysfluent patients’ language may mimic that of a patient with a thought disorder or intellectual disability, resulting in misdiagnosis.1
3. Meet the interpreter before the session. Discuss your goals and explain the meaning of psychiatric terms and symptoms to help him or her communicate your message and interpret the patient’s response.2
ASL is not visual English. Some concepts do not translate into ASL and need to be modified or omitted by interpreters. English idioms often used in diagnostic interviews, such as “feeling blue” or “feeling keyed up or on edge,” do not have exact ASL translations.
An interpreter has to use his or her judgment on how to translate these concepts in ASL. Some symptoms such as “panic attack” and “auditory hallucination” do not have corresponding signs in ASL.
Interpreters may convey these concepts by describing or even acting them out. Ambiguity is difficult to maintain in ASL. Interpreters often have to give examples or lists of possible choices to communicate a concept. Some open-ended questions must be transformed into multiple-choice questions, which can be leading or narrow potential responses.
4. Be aware of privacy. Certified interpreters are required to maintain confidentiality. However, a deaf patient might not have the same interpreter over the course of treatment. The deaf community is close-knit, and a patient using multiple interpreters may worry about confidentiality. Deaf people are likely to use interpreters in a variety of settings and may feel uncomfortable working with the same interpreter in a psychiatric setting and other social situations. The deaf patient always has final say over whether or not to work with a particular interpreter.
5. Avoid using families as interpreters. Even if they are fluent in ASL, family members can distort examination findings and will eliminate confidentiality. If a family member insists on serving as an interpreter, discuss his or her reasons. For example, some families use a form of idiosyncratic gestures often called “home signs” instead of ASL. In this case, you would need a family member present because an ASL interpreter would not be familiar with these. In other situations, you may have to educate family members about why a certified interpreter is more appropriate.
6. Don’t rely on written English. A typical deaf person of normal intelligence has a fourth-grade reading level. Self-report measures of symptoms often are written for an eighth-grade reading or higher level. In psychiatry, subtle nuances in communication are critical, and relying on written English could cause misunderstandings.
Do not use written notes passed back and forth with your patient to avoid the expense of an ASL interpreter. Physicians have been successfully sued for refusing to hire interpreters in the hope of “getting by” with written notes. In these cases the judgments against the physicians were not covered by malpractice insurance.
7. Don’t overpathologize. ASL is an expressive and dramatic language. In addition, deaf persons may have different personal space boundaries than hearing persons. Don’t mistake these cultural norms as evidence of a mood disorder or character pathology.
8. Be cautious when assessing a deaf person for psychosis and “hearing-based” phenomena such as auditory hallucinations. Psychotic disorders historically have been overdiagnosed in deaf patients when clinicians rely solely on subjective reports of symptoms. Instead, err on the side of caution unless you identify objective, observable evidence (such as bizarre behavior or clearly stated delusional beliefs).1
9. Clarify. Deaf persons—just like hearing persons—do not like to appear unknowledgeable. A patient may “nod along,” leading you to inaccurate conclusions and misdiagnosis. Use open-ended questions to elicit a full description of symptoms.
10. Be patient and plan ahead. Schedule longer sessions—as much as twice as long—to allow extra time for interpretation and double-checking comprehension. Remember that each question has to be asked, interpreted, answered, and interpreted again. Follow-up questions may be necessary to ensure comprehension.
1. Glickman N. Cognitive-behavioral therapy for deaf and hearing persons with language and learning challenges. New York, NY: Routledge; 2009.
2. Leigh IW, Corbett CA, Gutman V, et al. Providing psychological services to deaf individuals: a response to new perceptions of diversity. Prof Psychol Res Pr. 1996;4:364-371.
1. Glickman N. Cognitive-behavioral therapy for deaf and hearing persons with language and learning challenges. New York, NY: Routledge; 2009.
2. Leigh IW, Corbett CA, Gutman V, et al. Providing psychological services to deaf individuals: a response to new perceptions of diversity. Prof Psychol Res Pr. 1996;4:364-371.
PPD: 3 keys to assessing suicide risk
In the United States >33,000 people take their lives each year.1 Depression is involved in 65% to 90% of all suicides;2 however, some patients may not appear acutely depressed or might minimize suicidal thoughts to avoid treatment or hospitalization.
Having direct patient contact, information from collateral sources, and available medical records will guide you in developing a treatment plan, but also consider these 3 areas using the mnemonic PPD:
Past suicide attempts. One of the best predictors of future suicidal behavior is past attempts.3 Ask your patient if he or she has engaged in suicidal behaviors of increasing lethality, such as overdosing, cutting, or unintentional firearm injury. Patients who engage in “escalating attempts” are at a higher risk of harming themselves.4
Psychosis. Actively psychotic patients have difficulty contracting for safety. They may report hearing voices telling them to harm themselves or others. Ask patients about hallucinations and how they respond to these experiences even if the hallucinations do not involve suicidal content. For example, a patient with a delusion of having a deadly infectious disease may ingest an entire bottle of medication to eradicate the infection. This might seem like a suicide attempt, but the patient’s intent was not to die, but to “treat” himself or herself.
Drugs and alcohol. One-third of those who commit suicide test positive for alcohol and nearly 1 in 5 have evidence of opiates.5 Patients may abuse substances to regulate their moods; however, they are prone to suicidal behavior under the influence of drugs or alcohol. Ask your patient about substances he or she uses and how they impact suicidal thoughts.
Positive findings for ≥1 of the above criteria place a patient at higher risk for suicidal behavior.6 By incorporating these 3 factors into your suicide assessment, you will be better equipped to justify the level of care and treatment you recommend.
1. Centers for Disease Control and Prevention. Web-based Injury Statistics Query and Reporting System (WISQARS). Available at: http://cdc.gov/injury/wisqars. Accessed April 22, 2010.
2. Blumenthal SJ. Suicide: a guide to risk factors, assessment, and treatment of suicidal patients. Med Clin North Am. 1988;72:937-971.
3. Moscicki EK. Epidemiology of suicidal behavior. In: Silverman MM, Maris RW, eds. Suicide prevention: toward the year 2000. New York, NY: Guilford; 1985:22–35.
4. American Psychiatric Association Practice Guidelines for the Treatment of Psychiatric Disorders Compendium. Arlington, VA: American Psychiatric Association; 2004: 835–1027.
5. Karch DL, Dahlberg LL, Patel N, et al. Surveillance for violent deaths–national violent death reporting system, 16 states, 2006. MMWR Surveill Summ. 2009;58:1-44.
6. Oquendo MA, Malone KM, Ellis SP, et al. Inadequacy of antidepressant treatment for patients with major depression who are at risk for suicidal behavior. Am J Psychiatry. 1999;156:190-194.
In the United States >33,000 people take their lives each year.1 Depression is involved in 65% to 90% of all suicides;2 however, some patients may not appear acutely depressed or might minimize suicidal thoughts to avoid treatment or hospitalization.
Having direct patient contact, information from collateral sources, and available medical records will guide you in developing a treatment plan, but also consider these 3 areas using the mnemonic PPD:
Past suicide attempts. One of the best predictors of future suicidal behavior is past attempts.3 Ask your patient if he or she has engaged in suicidal behaviors of increasing lethality, such as overdosing, cutting, or unintentional firearm injury. Patients who engage in “escalating attempts” are at a higher risk of harming themselves.4
Psychosis. Actively psychotic patients have difficulty contracting for safety. They may report hearing voices telling them to harm themselves or others. Ask patients about hallucinations and how they respond to these experiences even if the hallucinations do not involve suicidal content. For example, a patient with a delusion of having a deadly infectious disease may ingest an entire bottle of medication to eradicate the infection. This might seem like a suicide attempt, but the patient’s intent was not to die, but to “treat” himself or herself.
Drugs and alcohol. One-third of those who commit suicide test positive for alcohol and nearly 1 in 5 have evidence of opiates.5 Patients may abuse substances to regulate their moods; however, they are prone to suicidal behavior under the influence of drugs or alcohol. Ask your patient about substances he or she uses and how they impact suicidal thoughts.
Positive findings for ≥1 of the above criteria place a patient at higher risk for suicidal behavior.6 By incorporating these 3 factors into your suicide assessment, you will be better equipped to justify the level of care and treatment you recommend.
In the United States >33,000 people take their lives each year.1 Depression is involved in 65% to 90% of all suicides;2 however, some patients may not appear acutely depressed or might minimize suicidal thoughts to avoid treatment or hospitalization.
Having direct patient contact, information from collateral sources, and available medical records will guide you in developing a treatment plan, but also consider these 3 areas using the mnemonic PPD:
Past suicide attempts. One of the best predictors of future suicidal behavior is past attempts.3 Ask your patient if he or she has engaged in suicidal behaviors of increasing lethality, such as overdosing, cutting, or unintentional firearm injury. Patients who engage in “escalating attempts” are at a higher risk of harming themselves.4
Psychosis. Actively psychotic patients have difficulty contracting for safety. They may report hearing voices telling them to harm themselves or others. Ask patients about hallucinations and how they respond to these experiences even if the hallucinations do not involve suicidal content. For example, a patient with a delusion of having a deadly infectious disease may ingest an entire bottle of medication to eradicate the infection. This might seem like a suicide attempt, but the patient’s intent was not to die, but to “treat” himself or herself.
Drugs and alcohol. One-third of those who commit suicide test positive for alcohol and nearly 1 in 5 have evidence of opiates.5 Patients may abuse substances to regulate their moods; however, they are prone to suicidal behavior under the influence of drugs or alcohol. Ask your patient about substances he or she uses and how they impact suicidal thoughts.
Positive findings for ≥1 of the above criteria place a patient at higher risk for suicidal behavior.6 By incorporating these 3 factors into your suicide assessment, you will be better equipped to justify the level of care and treatment you recommend.
1. Centers for Disease Control and Prevention. Web-based Injury Statistics Query and Reporting System (WISQARS). Available at: http://cdc.gov/injury/wisqars. Accessed April 22, 2010.
2. Blumenthal SJ. Suicide: a guide to risk factors, assessment, and treatment of suicidal patients. Med Clin North Am. 1988;72:937-971.
3. Moscicki EK. Epidemiology of suicidal behavior. In: Silverman MM, Maris RW, eds. Suicide prevention: toward the year 2000. New York, NY: Guilford; 1985:22–35.
4. American Psychiatric Association Practice Guidelines for the Treatment of Psychiatric Disorders Compendium. Arlington, VA: American Psychiatric Association; 2004: 835–1027.
5. Karch DL, Dahlberg LL, Patel N, et al. Surveillance for violent deaths–national violent death reporting system, 16 states, 2006. MMWR Surveill Summ. 2009;58:1-44.
6. Oquendo MA, Malone KM, Ellis SP, et al. Inadequacy of antidepressant treatment for patients with major depression who are at risk for suicidal behavior. Am J Psychiatry. 1999;156:190-194.
1. Centers for Disease Control and Prevention. Web-based Injury Statistics Query and Reporting System (WISQARS). Available at: http://cdc.gov/injury/wisqars. Accessed April 22, 2010.
2. Blumenthal SJ. Suicide: a guide to risk factors, assessment, and treatment of suicidal patients. Med Clin North Am. 1988;72:937-971.
3. Moscicki EK. Epidemiology of suicidal behavior. In: Silverman MM, Maris RW, eds. Suicide prevention: toward the year 2000. New York, NY: Guilford; 1985:22–35.
4. American Psychiatric Association Practice Guidelines for the Treatment of Psychiatric Disorders Compendium. Arlington, VA: American Psychiatric Association; 2004: 835–1027.
5. Karch DL, Dahlberg LL, Patel N, et al. Surveillance for violent deaths–national violent death reporting system, 16 states, 2006. MMWR Surveill Summ. 2009;58:1-44.
6. Oquendo MA, Malone KM, Ellis SP, et al. Inadequacy of antidepressant treatment for patients with major depression who are at risk for suicidal behavior. Am J Psychiatry. 1999;156:190-194.
Successfully navigating the 15-minute ‘med check’
How to reduce malpractice risk with better documentation.
Tips to make documentation easier, faster, and more satisfying” (Current Psychiatry, February 2008), I discussed documentation techniques at length. Table 3 reprints principles that may be especially helpful in practices that consist primarily of med checks.
Table 3
Keys to better documentation
| Technique | Benefits |
|---|---|
| Time and date your notes | After an adverse event, establish when you saw the patient, recorded findings, wrote orders, reviewed lab results, or discussed problems with others can make a big difference in how your care is viewed |
| Sooner is better | Charting completed long after an adverse event occurred is vulnerable to accusations of fabrication |
| Brief quotes | Verbatim statements (‘I’ve never considered suicide’) quickly convey key factors in your therapeutic decision |
| Dictate or use speech recognition software | You speak faster than you write allowing you to document more |
| Provide handouts | Patients often do not remember or understand much of medication instructions doctors tell them |
| Use rating scales | Record more information in a scientifically validated format |
| Try macros and templates | These reduce documentation time and help you remember to cover everything you should |
| Source: Adapted from reference 18 | |
Acknowledgment
Thanks to James Knoll IV, MD for his helpful input on this article.
1. Mojtabai R, Olfson M. National trends in psychotherapy by office-based psychiatrists. Arch Gen Psychiatry. 2008;65:962-970.
2. Lewis MH, Gohagan JK, Merenstein DJ. The locality rule and the physician’s dilemma: local medical practices vs the national standard of care. JAMA. 2007;297(23):2633-2637.
3. Gabbard GO. Deconstructing the “med check.” Psychiatric Times. September 3, 2009. Available at: http://www.psychiatrictimes.com/display/article/10168/1444238. Accessed April 28, 2010.
4. Pies RW. Psychiatrists, physicians, and the prescriptive bond. Psychiatric Times. April 16, 2010. Available at: http://www.psychiatrictimes.com/blog/couchincrisis/content/article/10168/1555057. Accessed April 28, 2010.
5. Carlat DJ. Unhinged: the trouble with psychiatry—a doctor’s revelations about a profession in crisis. New York, NY: Free Press; 2010.
6. Nemeroff CB. The myth of the med check in psychopharmacology. Presented at: Presidential Symposium, Annual Meeting of the American Psychiatric Association; May 7, 2008; Washington, DC.
7. Rush W, Gochfeli L, Minkov K, et al. Medication visits: visit time and quality—the connection. Compliance Watch. 2009;2(2):13-15.
8. Fine P. Psychodynamic psychiatry in community settings. J Am Acad Psychoanal Dyn Psychiatry. 2007;35:431-441.
9. Sherman C. Don’t forget therapeutic skills even during a “med check.” Clinical Psychiatry News. 2002;30(7):390.-Available at: http://findarticles.com/p/articles/mi_hb4345/is_7_30/ai_n28933329. Accessed April 28, 2010.
10. Ackerman SJ, Hilsenroth MJ. A review of therapist characteristics and techniques positively impacting the therapeutic alliance. Clinical Psychology Rev. 2003;23:1-33.
11. Guggenheim FG. Prime time: maximizing the therapeutic experience—a primer for psychiatric clinicians. New York, NY: Routledge; 2009.
12. Saks ER. The center cannot hold: my journey through madness. New York, NY: Hyperion; 2007.
13. Pincus HA, Tanielian TL, Marcus SC, et al. Prescribing trends in psychotropic medications: primary care, psychiatry, and other medical specialties. JAMA. 1998;279(7):526-531.
14. Harman JS, Veazie PJ, Lyness JM. Primary care physician office visits for depression by older Americans. J Gen Intern Med. 2006;21:926-930.
15. Chen LM, Farwell WR, Jha AK. Primary care visit duration and quality: does good care take longer? Arch Intern Med. 2009;169:1866-1872.
16. Gilchrist VJ, Stange KC, Flocke SA, et al. A comparison of the National Ambulatory Medical Care Survey (NAMCS) measurement approach with direct observation of outpatient visits. Med Care. 2004;42(3):276-280.
17. Moffic HS. Make the most of the “15-minute med-check.” Current Psychiatry. 2006;5(9):116.-
18. Mossman D. Tips to make documentation easier, faster, and more satisfying. Current Psychiatry. 2008;7(2):84-86.
How to reduce malpractice risk with better documentation.
Tips to make documentation easier, faster, and more satisfying” (Current Psychiatry, February 2008), I discussed documentation techniques at length. Table 3 reprints principles that may be especially helpful in practices that consist primarily of med checks.
Table 3
Keys to better documentation
| Technique | Benefits |
|---|---|
| Time and date your notes | After an adverse event, establish when you saw the patient, recorded findings, wrote orders, reviewed lab results, or discussed problems with others can make a big difference in how your care is viewed |
| Sooner is better | Charting completed long after an adverse event occurred is vulnerable to accusations of fabrication |
| Brief quotes | Verbatim statements (‘I’ve never considered suicide’) quickly convey key factors in your therapeutic decision |
| Dictate or use speech recognition software | You speak faster than you write allowing you to document more |
| Provide handouts | Patients often do not remember or understand much of medication instructions doctors tell them |
| Use rating scales | Record more information in a scientifically validated format |
| Try macros and templates | These reduce documentation time and help you remember to cover everything you should |
| Source: Adapted from reference 18 | |
Acknowledgment
Thanks to James Knoll IV, MD for his helpful input on this article.
How to reduce malpractice risk with better documentation.
Tips to make documentation easier, faster, and more satisfying” (Current Psychiatry, February 2008), I discussed documentation techniques at length. Table 3 reprints principles that may be especially helpful in practices that consist primarily of med checks.
Table 3
Keys to better documentation
| Technique | Benefits |
|---|---|
| Time and date your notes | After an adverse event, establish when you saw the patient, recorded findings, wrote orders, reviewed lab results, or discussed problems with others can make a big difference in how your care is viewed |
| Sooner is better | Charting completed long after an adverse event occurred is vulnerable to accusations of fabrication |
| Brief quotes | Verbatim statements (‘I’ve never considered suicide’) quickly convey key factors in your therapeutic decision |
| Dictate or use speech recognition software | You speak faster than you write allowing you to document more |
| Provide handouts | Patients often do not remember or understand much of medication instructions doctors tell them |
| Use rating scales | Record more information in a scientifically validated format |
| Try macros and templates | These reduce documentation time and help you remember to cover everything you should |
| Source: Adapted from reference 18 | |
Acknowledgment
Thanks to James Knoll IV, MD for his helpful input on this article.
1. Mojtabai R, Olfson M. National trends in psychotherapy by office-based psychiatrists. Arch Gen Psychiatry. 2008;65:962-970.
2. Lewis MH, Gohagan JK, Merenstein DJ. The locality rule and the physician’s dilemma: local medical practices vs the national standard of care. JAMA. 2007;297(23):2633-2637.
3. Gabbard GO. Deconstructing the “med check.” Psychiatric Times. September 3, 2009. Available at: http://www.psychiatrictimes.com/display/article/10168/1444238. Accessed April 28, 2010.
4. Pies RW. Psychiatrists, physicians, and the prescriptive bond. Psychiatric Times. April 16, 2010. Available at: http://www.psychiatrictimes.com/blog/couchincrisis/content/article/10168/1555057. Accessed April 28, 2010.
5. Carlat DJ. Unhinged: the trouble with psychiatry—a doctor’s revelations about a profession in crisis. New York, NY: Free Press; 2010.
6. Nemeroff CB. The myth of the med check in psychopharmacology. Presented at: Presidential Symposium, Annual Meeting of the American Psychiatric Association; May 7, 2008; Washington, DC.
7. Rush W, Gochfeli L, Minkov K, et al. Medication visits: visit time and quality—the connection. Compliance Watch. 2009;2(2):13-15.
8. Fine P. Psychodynamic psychiatry in community settings. J Am Acad Psychoanal Dyn Psychiatry. 2007;35:431-441.
9. Sherman C. Don’t forget therapeutic skills even during a “med check.” Clinical Psychiatry News. 2002;30(7):390.-Available at: http://findarticles.com/p/articles/mi_hb4345/is_7_30/ai_n28933329. Accessed April 28, 2010.
10. Ackerman SJ, Hilsenroth MJ. A review of therapist characteristics and techniques positively impacting the therapeutic alliance. Clinical Psychology Rev. 2003;23:1-33.
11. Guggenheim FG. Prime time: maximizing the therapeutic experience—a primer for psychiatric clinicians. New York, NY: Routledge; 2009.
12. Saks ER. The center cannot hold: my journey through madness. New York, NY: Hyperion; 2007.
13. Pincus HA, Tanielian TL, Marcus SC, et al. Prescribing trends in psychotropic medications: primary care, psychiatry, and other medical specialties. JAMA. 1998;279(7):526-531.
14. Harman JS, Veazie PJ, Lyness JM. Primary care physician office visits for depression by older Americans. J Gen Intern Med. 2006;21:926-930.
15. Chen LM, Farwell WR, Jha AK. Primary care visit duration and quality: does good care take longer? Arch Intern Med. 2009;169:1866-1872.
16. Gilchrist VJ, Stange KC, Flocke SA, et al. A comparison of the National Ambulatory Medical Care Survey (NAMCS) measurement approach with direct observation of outpatient visits. Med Care. 2004;42(3):276-280.
17. Moffic HS. Make the most of the “15-minute med-check.” Current Psychiatry. 2006;5(9):116.-
18. Mossman D. Tips to make documentation easier, faster, and more satisfying. Current Psychiatry. 2008;7(2):84-86.
1. Mojtabai R, Olfson M. National trends in psychotherapy by office-based psychiatrists. Arch Gen Psychiatry. 2008;65:962-970.
2. Lewis MH, Gohagan JK, Merenstein DJ. The locality rule and the physician’s dilemma: local medical practices vs the national standard of care. JAMA. 2007;297(23):2633-2637.
3. Gabbard GO. Deconstructing the “med check.” Psychiatric Times. September 3, 2009. Available at: http://www.psychiatrictimes.com/display/article/10168/1444238. Accessed April 28, 2010.
4. Pies RW. Psychiatrists, physicians, and the prescriptive bond. Psychiatric Times. April 16, 2010. Available at: http://www.psychiatrictimes.com/blog/couchincrisis/content/article/10168/1555057. Accessed April 28, 2010.
5. Carlat DJ. Unhinged: the trouble with psychiatry—a doctor’s revelations about a profession in crisis. New York, NY: Free Press; 2010.
6. Nemeroff CB. The myth of the med check in psychopharmacology. Presented at: Presidential Symposium, Annual Meeting of the American Psychiatric Association; May 7, 2008; Washington, DC.
7. Rush W, Gochfeli L, Minkov K, et al. Medication visits: visit time and quality—the connection. Compliance Watch. 2009;2(2):13-15.
8. Fine P. Psychodynamic psychiatry in community settings. J Am Acad Psychoanal Dyn Psychiatry. 2007;35:431-441.
9. Sherman C. Don’t forget therapeutic skills even during a “med check.” Clinical Psychiatry News. 2002;30(7):390.-Available at: http://findarticles.com/p/articles/mi_hb4345/is_7_30/ai_n28933329. Accessed April 28, 2010.
10. Ackerman SJ, Hilsenroth MJ. A review of therapist characteristics and techniques positively impacting the therapeutic alliance. Clinical Psychology Rev. 2003;23:1-33.
11. Guggenheim FG. Prime time: maximizing the therapeutic experience—a primer for psychiatric clinicians. New York, NY: Routledge; 2009.
12. Saks ER. The center cannot hold: my journey through madness. New York, NY: Hyperion; 2007.
13. Pincus HA, Tanielian TL, Marcus SC, et al. Prescribing trends in psychotropic medications: primary care, psychiatry, and other medical specialties. JAMA. 1998;279(7):526-531.
14. Harman JS, Veazie PJ, Lyness JM. Primary care physician office visits for depression by older Americans. J Gen Intern Med. 2006;21:926-930.
15. Chen LM, Farwell WR, Jha AK. Primary care visit duration and quality: does good care take longer? Arch Intern Med. 2009;169:1866-1872.
16. Gilchrist VJ, Stange KC, Flocke SA, et al. A comparison of the National Ambulatory Medical Care Survey (NAMCS) measurement approach with direct observation of outpatient visits. Med Care. 2004;42(3):276-280.
17. Moffic HS. Make the most of the “15-minute med-check.” Current Psychiatry. 2006;5(9):116.-
18. Mossman D. Tips to make documentation easier, faster, and more satisfying. Current Psychiatry. 2008;7(2):84-86.
Cutoff points for treatment
In “Children with tic disorders: How to match treatment with symptoms” (Current Psychiatry, March 2010), the authors describe Sammy, who developed symptoms of Tourette syndrome. However, we should not forget that children with allergies often experience itching or irritation of the eyelids, nostrils, and throat that causes them to blink, sniff, touch their nose, or clear their throat. The presence of sneezing, runny, or congested nose may help distinguish allergies, but there is an interesting overlap.
I was intrigued by the authors’ suggestion to tell parents “you typically do not treat children with antipsychotics for more than one year continuously.” I thought the decision to continue or discontinue depended on an individualized risk and benefit approach. Are the authors referring to Tourette syndrome specifically or all indications including psychosis and/or mania?
Ian T. Webber, MD
Community Psychiatry Associates
San Francisco, CA
The authors respond
Children with allergies may receive a diagnosis of a tic disorder, but it has been our experience that the tic disorder initially is mislabeled as an allergy. We recommend asking about premonitory urge or sensation, whether the symptom can be temporarily suppressed, and whether there is a sense of relief after the action. Noting a seasonal pattern also may help make the correct diagnosis.
We recommend not planning antipsychotic treatment for longer than 1 year because the risks of metabolic side effects may outweigh benefit for a tic disorder with a waxing and waning course. The decision to continue medication is based on the individual’s risk-benefit ratio. Our article focused on treatment of tic disorders; psychosis and mania are extremely rare in children. If a child presents with out-of-control behavior, we recommend a behavior plan and parental guidance over antipsychotics if possible. Short-term antipsychotic use may be indicated, although most are off-label in the pediatric population. Tapering the medication periodically to determine whether it is still necessary can help limit side effects, such as diabetes.
Jeste et al1 note that with each year of antipsychotic use, 5% of patients will show signs of tardive dyskinesia. In a retrospective study of 60 adolescents treated with risperidone, typical antipsychotics, or no antipsychotic, the risperidone-treated group gained significantly more body mass than those receiving a conventional antipsychotic over 6 months.2 Degrauw et al3 reported that weight gain is not always excessive in pediatric patients treated with antipsychotics for >1 year.
Elena Harris, MD, PhD
Assistant professor
Division of child and adolescent psychiatry
Steve W. Wu, MD
Assistant professor
Division of child neurology
Cincinnati Children’s Hospital Medical Center
Cincinnati, OH
1. Jeste DV, Caligiuri MP. Tardive dyskinesia. Schizophr Bull. 1993;19:303-315.
2. Kelly DL, Conley RR, Love RC, et al. Weight gain in adolescents treated with risperidone and conventional antipsychotics over six months. J Child Adolesc Psychopharmacol. 1998;8(3):151-159.
3. Degrauw RS, Li JZ, Gilbert DL. Body mass index changes and chronic neuroleptic drug treatment for Tourette syndrome. Pediatr Neurol. 2009;41:183-186.
In “Children with tic disorders: How to match treatment with symptoms” (Current Psychiatry, March 2010), the authors describe Sammy, who developed symptoms of Tourette syndrome. However, we should not forget that children with allergies often experience itching or irritation of the eyelids, nostrils, and throat that causes them to blink, sniff, touch their nose, or clear their throat. The presence of sneezing, runny, or congested nose may help distinguish allergies, but there is an interesting overlap.
I was intrigued by the authors’ suggestion to tell parents “you typically do not treat children with antipsychotics for more than one year continuously.” I thought the decision to continue or discontinue depended on an individualized risk and benefit approach. Are the authors referring to Tourette syndrome specifically or all indications including psychosis and/or mania?
Ian T. Webber, MD
Community Psychiatry Associates
San Francisco, CA
The authors respond
Children with allergies may receive a diagnosis of a tic disorder, but it has been our experience that the tic disorder initially is mislabeled as an allergy. We recommend asking about premonitory urge or sensation, whether the symptom can be temporarily suppressed, and whether there is a sense of relief after the action. Noting a seasonal pattern also may help make the correct diagnosis.
We recommend not planning antipsychotic treatment for longer than 1 year because the risks of metabolic side effects may outweigh benefit for a tic disorder with a waxing and waning course. The decision to continue medication is based on the individual’s risk-benefit ratio. Our article focused on treatment of tic disorders; psychosis and mania are extremely rare in children. If a child presents with out-of-control behavior, we recommend a behavior plan and parental guidance over antipsychotics if possible. Short-term antipsychotic use may be indicated, although most are off-label in the pediatric population. Tapering the medication periodically to determine whether it is still necessary can help limit side effects, such as diabetes.
Jeste et al1 note that with each year of antipsychotic use, 5% of patients will show signs of tardive dyskinesia. In a retrospective study of 60 adolescents treated with risperidone, typical antipsychotics, or no antipsychotic, the risperidone-treated group gained significantly more body mass than those receiving a conventional antipsychotic over 6 months.2 Degrauw et al3 reported that weight gain is not always excessive in pediatric patients treated with antipsychotics for >1 year.
Elena Harris, MD, PhD
Assistant professor
Division of child and adolescent psychiatry
Steve W. Wu, MD
Assistant professor
Division of child neurology
Cincinnati Children’s Hospital Medical Center
Cincinnati, OH
In “Children with tic disorders: How to match treatment with symptoms” (Current Psychiatry, March 2010), the authors describe Sammy, who developed symptoms of Tourette syndrome. However, we should not forget that children with allergies often experience itching or irritation of the eyelids, nostrils, and throat that causes them to blink, sniff, touch their nose, or clear their throat. The presence of sneezing, runny, or congested nose may help distinguish allergies, but there is an interesting overlap.
I was intrigued by the authors’ suggestion to tell parents “you typically do not treat children with antipsychotics for more than one year continuously.” I thought the decision to continue or discontinue depended on an individualized risk and benefit approach. Are the authors referring to Tourette syndrome specifically or all indications including psychosis and/or mania?
Ian T. Webber, MD
Community Psychiatry Associates
San Francisco, CA
The authors respond
Children with allergies may receive a diagnosis of a tic disorder, but it has been our experience that the tic disorder initially is mislabeled as an allergy. We recommend asking about premonitory urge or sensation, whether the symptom can be temporarily suppressed, and whether there is a sense of relief after the action. Noting a seasonal pattern also may help make the correct diagnosis.
We recommend not planning antipsychotic treatment for longer than 1 year because the risks of metabolic side effects may outweigh benefit for a tic disorder with a waxing and waning course. The decision to continue medication is based on the individual’s risk-benefit ratio. Our article focused on treatment of tic disorders; psychosis and mania are extremely rare in children. If a child presents with out-of-control behavior, we recommend a behavior plan and parental guidance over antipsychotics if possible. Short-term antipsychotic use may be indicated, although most are off-label in the pediatric population. Tapering the medication periodically to determine whether it is still necessary can help limit side effects, such as diabetes.
Jeste et al1 note that with each year of antipsychotic use, 5% of patients will show signs of tardive dyskinesia. In a retrospective study of 60 adolescents treated with risperidone, typical antipsychotics, or no antipsychotic, the risperidone-treated group gained significantly more body mass than those receiving a conventional antipsychotic over 6 months.2 Degrauw et al3 reported that weight gain is not always excessive in pediatric patients treated with antipsychotics for >1 year.
Elena Harris, MD, PhD
Assistant professor
Division of child and adolescent psychiatry
Steve W. Wu, MD
Assistant professor
Division of child neurology
Cincinnati Children’s Hospital Medical Center
Cincinnati, OH
1. Jeste DV, Caligiuri MP. Tardive dyskinesia. Schizophr Bull. 1993;19:303-315.
2. Kelly DL, Conley RR, Love RC, et al. Weight gain in adolescents treated with risperidone and conventional antipsychotics over six months. J Child Adolesc Psychopharmacol. 1998;8(3):151-159.
3. Degrauw RS, Li JZ, Gilbert DL. Body mass index changes and chronic neuroleptic drug treatment for Tourette syndrome. Pediatr Neurol. 2009;41:183-186.
1. Jeste DV, Caligiuri MP. Tardive dyskinesia. Schizophr Bull. 1993;19:303-315.
2. Kelly DL, Conley RR, Love RC, et al. Weight gain in adolescents treated with risperidone and conventional antipsychotics over six months. J Child Adolesc Psychopharmacol. 1998;8(3):151-159.
3. Degrauw RS, Li JZ, Gilbert DL. Body mass index changes and chronic neuroleptic drug treatment for Tourette syndrome. Pediatr Neurol. 2009;41:183-186.
A unifying manifesto
Dr. Henry A. Nasrallah’s “A psychiatric manifesto” (From the Editor, Current Psychiatry, April 2010; available at http://bit.ly/9tOuvi) is a precise description of the beauty and hardships unique to our ever-evolving specialty. I am proud to share this description with medical students, psychiatry residents, and fellows and urge others to consider doing the same. I find myself reflecting on each of these core themes during the day. It is true that as we stand on the brink of great neuroscience discoveries with potential to benefit millions of our patients, we simultaneously face “more detractors and self-appointed critics than any other medical specialty.” I am hopeful that this living manifesto will bolster collective adhesion within our field while also helping to educate non-psychiatrists of “who we are and what we do.”
James G. MacKenzie, DO
Medical director, consultation and emergency services
Department of child and adolescent psychiatry
Children’s Memorial Hospital
Chicago, IL
Dr. Henry A. Nasrallah’s “A psychiatric manifesto” (From the Editor, Current Psychiatry, April 2010; available at http://bit.ly/9tOuvi) is a precise description of the beauty and hardships unique to our ever-evolving specialty. I am proud to share this description with medical students, psychiatry residents, and fellows and urge others to consider doing the same. I find myself reflecting on each of these core themes during the day. It is true that as we stand on the brink of great neuroscience discoveries with potential to benefit millions of our patients, we simultaneously face “more detractors and self-appointed critics than any other medical specialty.” I am hopeful that this living manifesto will bolster collective adhesion within our field while also helping to educate non-psychiatrists of “who we are and what we do.”
James G. MacKenzie, DO
Medical director, consultation and emergency services
Department of child and adolescent psychiatry
Children’s Memorial Hospital
Chicago, IL
Dr. Henry A. Nasrallah’s “A psychiatric manifesto” (From the Editor, Current Psychiatry, April 2010; available at http://bit.ly/9tOuvi) is a precise description of the beauty and hardships unique to our ever-evolving specialty. I am proud to share this description with medical students, psychiatry residents, and fellows and urge others to consider doing the same. I find myself reflecting on each of these core themes during the day. It is true that as we stand on the brink of great neuroscience discoveries with potential to benefit millions of our patients, we simultaneously face “more detractors and self-appointed critics than any other medical specialty.” I am hopeful that this living manifesto will bolster collective adhesion within our field while also helping to educate non-psychiatrists of “who we are and what we do.”
James G. MacKenzie, DO
Medical director, consultation and emergency services
Department of child and adolescent psychiatry
Children’s Memorial Hospital
Chicago, IL
A psychiatrist/lawyer crossfire
Do lawyers understand psychiatry? To answer that semi-rhetorical question, I imagined the following conversation between 2 friends, Barry the barrister and Harry the psychiatrist.
Barry: Harry, I think psychiatry is a politically incorrect discipline.
Harry: How so, my dear friend?
Barry: Well, psychiatrists hospitalize people against their will, strip them of their civil liberties, and force them to take powerful, mind-altering drugs.
Harry: Barry, when you think about it objectively, involuntary hospitalization is a compassionate and legal act for people suffering from a brain disease that makes them suicidal or homicidal and a danger to themselves and others with no insight that they are sick. Once treated and improved, patients regain their civil liberties and often thank us for providing care against their will. And a person needs a healthy brain to properly exercise one’s civil liberties.
Barry: What about electroconvulsive therapy (ECT)? Why would you subject people to such a primitive procedure?
Harry: For your information, when used for treating severely depressed persons who do not respond to multiple medications, ECT is one of the most effective procedures.1 It saves the lives of suicidal patients and restores their functioning. Published studies show that ECT stimulates neurogenesis and helps regenerate brain tissue by stimulating the production of new brain cells in the hippocampus, a critical structure that loses tissue during depression.2 Other neurostimulation techniques, including repetitive transcranial magnetic stimulation (rTMS) and vagus nerve stimulation (VNS), recently have been FDA-approved for severe depression. Deep brain stimulation (DBS) also is emerging as a promising treatment.3
Barry: But Harry, you psychiatrists medicalize normal emotions such as sadness or grief and pathologize high energy in children, transforming them into diagnostic labels like attention-deficit/hyperactivity disorder.
Harry: Normal sadness or grief is not a psychiatric diagnosis. People seek psychiatric help on their own or are brought in by their loved ones when an emotional tsunami devastates their lives, causing them to stop functioning, demonstrate unusual behavior, or express suicidal thoughts. Children referred for treatment because of uncontrollable behaviors at home and school are afflicted by neurobiologic disorders and FDA-approved medications help them to lead normal lives. Do you really believe children are incapable of having a psychiatric illness?
Barry: OK, but isn’t it irresponsible for psychiatrists to administer powerful antipsychotics to elderly persons in nursing homes when the FDA has issued a “black-box” warning that this practice may be fatal?
Harry: I wish that one day you would visit a facility for older persons with Alzheimer’s disease to see that as many as half of these individuals become psychotic, extremely agitated and assaultive, or a risk to themselves, the staff, and other patients.4 Would you like to advise physicians about how to manage serious dementia-related delusions and hallucinations without using antipsychotics?
Barry: You keep throwing the ball in my court, but psychiatrists violate the law daily by using psychiatric medications for conditions other than their approved use. Some studies show that up to 60% of psychotropic drugs are being used off-label.5
Harry: Less than 20% of DSM-IV diagnoses have an FDA-approved medication.6 What do you expect psychiatrists to tell the other 80%—“Sorry, come back in a decade or 2 when a drug is finally developed and approved for your condition”? Psychiatrists are compassionate when they select a medication the FDA has already deemed safe for a certain psychiatric illness and prescribe it for other ailments that have some of the same symptoms. Thanks to judiciously implemented off-label practices, many patients obtain some relief instead of continuing to suffer. And there is no law against off-label prescribing. It is left up to the physician’s discretion to use existing tools when there are no other options.
Barry: Harry, your responses have led me to view psychiatry more positively. I urge you write an editorial in a legal journal to debunk the myths.
Harry: Thanks for the suggestion, Barry. I think I will write an editorial in a widely read psychiatry journal called Current Psychiatry and avail it to all attorneys via CurrentPsychiatry.com.
1. Fink M. Electroconvulsive therapy. 2nd ed. New York, NY: Oxford University Press; 2008.
2. Bolwig TG, Madsen TM. Electroconvulsive therapy in melancholia: the role of hippocampal neurogenesis. Acta Psychiatr Scand Suppl. 2007;433:130-135.
3. Kuhn J, Gründler TO, Lenartz D, et al. Deep brain stimulation for psychiatric disorders. Dtsch Arztebl Int. 2010;107:105-113.
4. Chahine LM, Acar D, Chemali Z. The elderly safety imperative and antipsychotic usage. Harv Rev Psychiatry. 2010;18(3):158-172.
5. Chen H, Reeves JH, Fincham JE, et al. Off-label use of antidepressant, anticonvulsant, and antipsychotic medications among Georgia medicaid enrollees in 2001. J Clin Psychiatry. 2006;67(6):972-982.
6. Devulapalli K, Nasrallah HA. An analysis of the high psychotropic off-label use in psychiatric disorders: the majority of psychiatric diagnoses have no approved drug. Asian Journal of Psychiatry. 2009;2:29-36.
Do lawyers understand psychiatry? To answer that semi-rhetorical question, I imagined the following conversation between 2 friends, Barry the barrister and Harry the psychiatrist.
Barry: Harry, I think psychiatry is a politically incorrect discipline.
Harry: How so, my dear friend?
Barry: Well, psychiatrists hospitalize people against their will, strip them of their civil liberties, and force them to take powerful, mind-altering drugs.
Harry: Barry, when you think about it objectively, involuntary hospitalization is a compassionate and legal act for people suffering from a brain disease that makes them suicidal or homicidal and a danger to themselves and others with no insight that they are sick. Once treated and improved, patients regain their civil liberties and often thank us for providing care against their will. And a person needs a healthy brain to properly exercise one’s civil liberties.
Barry: What about electroconvulsive therapy (ECT)? Why would you subject people to such a primitive procedure?
Harry: For your information, when used for treating severely depressed persons who do not respond to multiple medications, ECT is one of the most effective procedures.1 It saves the lives of suicidal patients and restores their functioning. Published studies show that ECT stimulates neurogenesis and helps regenerate brain tissue by stimulating the production of new brain cells in the hippocampus, a critical structure that loses tissue during depression.2 Other neurostimulation techniques, including repetitive transcranial magnetic stimulation (rTMS) and vagus nerve stimulation (VNS), recently have been FDA-approved for severe depression. Deep brain stimulation (DBS) also is emerging as a promising treatment.3
Barry: But Harry, you psychiatrists medicalize normal emotions such as sadness or grief and pathologize high energy in children, transforming them into diagnostic labels like attention-deficit/hyperactivity disorder.
Harry: Normal sadness or grief is not a psychiatric diagnosis. People seek psychiatric help on their own or are brought in by their loved ones when an emotional tsunami devastates their lives, causing them to stop functioning, demonstrate unusual behavior, or express suicidal thoughts. Children referred for treatment because of uncontrollable behaviors at home and school are afflicted by neurobiologic disorders and FDA-approved medications help them to lead normal lives. Do you really believe children are incapable of having a psychiatric illness?
Barry: OK, but isn’t it irresponsible for psychiatrists to administer powerful antipsychotics to elderly persons in nursing homes when the FDA has issued a “black-box” warning that this practice may be fatal?
Harry: I wish that one day you would visit a facility for older persons with Alzheimer’s disease to see that as many as half of these individuals become psychotic, extremely agitated and assaultive, or a risk to themselves, the staff, and other patients.4 Would you like to advise physicians about how to manage serious dementia-related delusions and hallucinations without using antipsychotics?
Barry: You keep throwing the ball in my court, but psychiatrists violate the law daily by using psychiatric medications for conditions other than their approved use. Some studies show that up to 60% of psychotropic drugs are being used off-label.5
Harry: Less than 20% of DSM-IV diagnoses have an FDA-approved medication.6 What do you expect psychiatrists to tell the other 80%—“Sorry, come back in a decade or 2 when a drug is finally developed and approved for your condition”? Psychiatrists are compassionate when they select a medication the FDA has already deemed safe for a certain psychiatric illness and prescribe it for other ailments that have some of the same symptoms. Thanks to judiciously implemented off-label practices, many patients obtain some relief instead of continuing to suffer. And there is no law against off-label prescribing. It is left up to the physician’s discretion to use existing tools when there are no other options.
Barry: Harry, your responses have led me to view psychiatry more positively. I urge you write an editorial in a legal journal to debunk the myths.
Harry: Thanks for the suggestion, Barry. I think I will write an editorial in a widely read psychiatry journal called Current Psychiatry and avail it to all attorneys via CurrentPsychiatry.com.
Do lawyers understand psychiatry? To answer that semi-rhetorical question, I imagined the following conversation between 2 friends, Barry the barrister and Harry the psychiatrist.
Barry: Harry, I think psychiatry is a politically incorrect discipline.
Harry: How so, my dear friend?
Barry: Well, psychiatrists hospitalize people against their will, strip them of their civil liberties, and force them to take powerful, mind-altering drugs.
Harry: Barry, when you think about it objectively, involuntary hospitalization is a compassionate and legal act for people suffering from a brain disease that makes them suicidal or homicidal and a danger to themselves and others with no insight that they are sick. Once treated and improved, patients regain their civil liberties and often thank us for providing care against their will. And a person needs a healthy brain to properly exercise one’s civil liberties.
Barry: What about electroconvulsive therapy (ECT)? Why would you subject people to such a primitive procedure?
Harry: For your information, when used for treating severely depressed persons who do not respond to multiple medications, ECT is one of the most effective procedures.1 It saves the lives of suicidal patients and restores their functioning. Published studies show that ECT stimulates neurogenesis and helps regenerate brain tissue by stimulating the production of new brain cells in the hippocampus, a critical structure that loses tissue during depression.2 Other neurostimulation techniques, including repetitive transcranial magnetic stimulation (rTMS) and vagus nerve stimulation (VNS), recently have been FDA-approved for severe depression. Deep brain stimulation (DBS) also is emerging as a promising treatment.3
Barry: But Harry, you psychiatrists medicalize normal emotions such as sadness or grief and pathologize high energy in children, transforming them into diagnostic labels like attention-deficit/hyperactivity disorder.
Harry: Normal sadness or grief is not a psychiatric diagnosis. People seek psychiatric help on their own or are brought in by their loved ones when an emotional tsunami devastates their lives, causing them to stop functioning, demonstrate unusual behavior, or express suicidal thoughts. Children referred for treatment because of uncontrollable behaviors at home and school are afflicted by neurobiologic disorders and FDA-approved medications help them to lead normal lives. Do you really believe children are incapable of having a psychiatric illness?
Barry: OK, but isn’t it irresponsible for psychiatrists to administer powerful antipsychotics to elderly persons in nursing homes when the FDA has issued a “black-box” warning that this practice may be fatal?
Harry: I wish that one day you would visit a facility for older persons with Alzheimer’s disease to see that as many as half of these individuals become psychotic, extremely agitated and assaultive, or a risk to themselves, the staff, and other patients.4 Would you like to advise physicians about how to manage serious dementia-related delusions and hallucinations without using antipsychotics?
Barry: You keep throwing the ball in my court, but psychiatrists violate the law daily by using psychiatric medications for conditions other than their approved use. Some studies show that up to 60% of psychotropic drugs are being used off-label.5
Harry: Less than 20% of DSM-IV diagnoses have an FDA-approved medication.6 What do you expect psychiatrists to tell the other 80%—“Sorry, come back in a decade or 2 when a drug is finally developed and approved for your condition”? Psychiatrists are compassionate when they select a medication the FDA has already deemed safe for a certain psychiatric illness and prescribe it for other ailments that have some of the same symptoms. Thanks to judiciously implemented off-label practices, many patients obtain some relief instead of continuing to suffer. And there is no law against off-label prescribing. It is left up to the physician’s discretion to use existing tools when there are no other options.
Barry: Harry, your responses have led me to view psychiatry more positively. I urge you write an editorial in a legal journal to debunk the myths.
Harry: Thanks for the suggestion, Barry. I think I will write an editorial in a widely read psychiatry journal called Current Psychiatry and avail it to all attorneys via CurrentPsychiatry.com.
1. Fink M. Electroconvulsive therapy. 2nd ed. New York, NY: Oxford University Press; 2008.
2. Bolwig TG, Madsen TM. Electroconvulsive therapy in melancholia: the role of hippocampal neurogenesis. Acta Psychiatr Scand Suppl. 2007;433:130-135.
3. Kuhn J, Gründler TO, Lenartz D, et al. Deep brain stimulation for psychiatric disorders. Dtsch Arztebl Int. 2010;107:105-113.
4. Chahine LM, Acar D, Chemali Z. The elderly safety imperative and antipsychotic usage. Harv Rev Psychiatry. 2010;18(3):158-172.
5. Chen H, Reeves JH, Fincham JE, et al. Off-label use of antidepressant, anticonvulsant, and antipsychotic medications among Georgia medicaid enrollees in 2001. J Clin Psychiatry. 2006;67(6):972-982.
6. Devulapalli K, Nasrallah HA. An analysis of the high psychotropic off-label use in psychiatric disorders: the majority of psychiatric diagnoses have no approved drug. Asian Journal of Psychiatry. 2009;2:29-36.
1. Fink M. Electroconvulsive therapy. 2nd ed. New York, NY: Oxford University Press; 2008.
2. Bolwig TG, Madsen TM. Electroconvulsive therapy in melancholia: the role of hippocampal neurogenesis. Acta Psychiatr Scand Suppl. 2007;433:130-135.
3. Kuhn J, Gründler TO, Lenartz D, et al. Deep brain stimulation for psychiatric disorders. Dtsch Arztebl Int. 2010;107:105-113.
4. Chahine LM, Acar D, Chemali Z. The elderly safety imperative and antipsychotic usage. Harv Rev Psychiatry. 2010;18(3):158-172.
5. Chen H, Reeves JH, Fincham JE, et al. Off-label use of antidepressant, anticonvulsant, and antipsychotic medications among Georgia medicaid enrollees in 2001. J Clin Psychiatry. 2006;67(6):972-982.
6. Devulapalli K, Nasrallah HA. An analysis of the high psychotropic off-label use in psychiatric disorders: the majority of psychiatric diagnoses have no approved drug. Asian Journal of Psychiatry. 2009;2:29-36.
The manipulative self-harmer
CASE: Self-destructive behaviors
After being acquitted of 4 counts of second-degree forgery for writing checks from her mother’s bank account, Ms. L, age 52, is sent to the state hospital for a forensic examination to determine competency. Two years later she is granted conditional release from the hospital, transferred to our not-for-profit community mental health center, and enrolled in an intensive inpatient treatment program to monitor forensic patients. She is legally required to comply with treatment recommendations.
At admission, Ms. L is diagnosed with major depression, recurrent, and borderline personality disorder (BPD). She has no history of antisocial behavior or criminal acts other than forging checks and has never spent time in prison, which makes it unlikely she has co morbid antisocial personality disorder (Table 1).1
Over the next 5 years Ms. L tests limits with the treatment team and acts out by engaging in self-harming behaviors. In 1 instance, she cuts her forearm deeply, stuffs the wound with mayonnaise and paper towels, and wraps her arm with a bandage. She wears a long-sleeved shirt to hide her wound, which is not discovered until a severe infection develops.
Ms. L has difficulty with coping skills and interpersonal relationships. She approaches others with ambivalence and mistrust and consistently expects them to demean or take advantage of her. Ms. L is manipulative, at times injuring herself after perceived wrongdoings by staff. For example, after her therapist reschedules a meeting because of an emergency, Ms. L pours scalding water on her foot.
Table 1
Cluster B personality disorders: Differential diagnosis
| Diagnosis | Features |
|---|---|
| Borderline personality disorder | Self-destructiveness, angry disruptions in close relationships, and chronic feelings of deep emptiness and loneliness |
| Histrionic personality disorder | Attention seeking, manipulative behavior, and rapidly shifting emotions |
| Antisocial personality disorder | Manipulative to gain profit, power, or other material gratification |
| Source: Reference 1 | |
The authors’ observations
Ms. L consistently displays 3 common constructs of BPD:
- primitive defense mechanisms
- identity diffusion
- generally intact reality testing.2
Defense mechanisms are psychological attempts to deal with intrapsychic stress. Splitting—vacillating between extremes of idealization and devaluation—is a fundamental primitive defense mechanism that is the root of BPD.2 Identity diffusion causes confusion about life goals and values and feelings of boredom and emptiness. This internal world leads a patient to have the same perception of the external world, which explains many symptoms of BPD, such as rapidly shifting moods, intense anger, lack of clear sense of self, fear of abandonment, and unstable and intense interpersonal relationships.2
Early in treatment, Ms. L had difficulty breaking a cycle of self-defeating behavior, such as destroying personal items, trying to hang herself, and gluing an ear plug in her ear. During an argument with a staff member, Ms. L punched a wall and fractured her left hand. BPD patients sometimes will “up the ante” when acting out. For example, one of our patients claimed to have planted a bomb in an elementary school and another swallowed inedible objects, including spoons, forks, and butter knives. In Ms. L’s case, we addressed her self-harm behavior by helping her:
- develop less destructive coping skills such as drawing or painting
- identify irrational thoughts that contribute to self harm.
HISTORY: Troubled past
Raised by her biologic parents, Ms. L met all developmental milestones. She denies a history of childhood abuse but reports experiencing “depression and memory loss” and relationship problems with her parents during adolescence. As a child she often missed school because she “did not want anyone to know what a disgusting person I was” and “I should have my head cut open and cut into little pieces for thinking such mean thoughts.” Ms. L dropped out of school in the twelfth grade but obtained her general educational development certificate.
Notes and letters Ms. L wrote while in treatment consistently refer to her negative self-image. Ms. L writes that she feels she does not deserve to “be a part of this world,” is “never good enough for anyone,” and “should be thrown away with the garbage.”
Ms. L vacillates between desiring a closer relationship with her parents, especially her mother, and wanting to “cut them out of my life for good.” She has minimal contact with her older sister. Ms. L is divorced and has 2 adult sons. She was involved sporadically in her sons’ lives when they were children, but now has no contact with them.
BPD and crime
Ms. L is enrolled in the “911 program,” which monitors individuals who have been found not guilty by reason of mental defect. Individuals with BPD often are convicted of serious and violent crimes, which may be because of BPD features such as interpersonal hostility and self-harm. Impulsivity, substance abuse, and parental neglect—all of which are associated with BPD—can increase risk of criminality.3 There is no evidence to suggest a direct link between BPD and criminality; however, over-representation of BPD in prison populations suggest that in severe cases it may increase criminogenic risk.1,3
TREATMENT: Worsened depression
When Ms. L arrives at our facility, her medication regimen includes fluoxetine, 80 mg/d, risperidone, 2 mg/d, and buspirone, 20 mg/d. Risperidone and buspirone are discontinued because of perceived lack of efficacy. Venlafaxine XR is added and titrated to 300 mg/d, and Ms. L receives lorazepam, 1 and 2 mg as needed. However, lorazepam carries risks because impulsivity and impaired judgment—which are common in BPD—can lead to dependence and abuse. We feel that in a supervised setting the risks can be managed.
Recently, staff witnessed Ms. L experiencing an episode that appeared to be a grand mal seizure. After Ms. L is evaluated at the local emergency room, her EEG is normal, but a neurologic consult recommends discontinuing fluoxetine or venlafaxine XR because they may have contributed to the seizure. We taper and discontinue venlafaxine XR but Ms. L complains bitterly that she is getting increasingly depressed. On several occasions she attempts to pit team members against each other.
Ms. L falls, injures her back, and begins to abuse opiates. After her prescription runs out, she obtains more from an intellectually limited patient in her treatment program. Ms. L says she is getting more depressed, threatens suicide, and is placed in a more restrictive in-patient setting. We consider adding pregabalin to address her pain and help with anxiety and impulse control but the consulting neurologist prescribes carbamazepine, 400 mg/d, and her pain improves.5,6
The authors’ observations
BPD treatment primarily is psychotherapeutic and emphasizes skill building (Table 2) with focused, symptom-targeted pharmacotherapy as indicated.4 Pharmacotherapy typically targets 3 domains:
- affective dysregulation
- impulsive-behavioral dyscontrol symptoms
- cognitive-perceptual symptoms.
Patients with prominent anxiety may benefit from benzodiazepines, although research on these agents for BPD is limited. Recent studies show efficacy with fluoxetine, olanzapine, or a combination of both,7 and divalproex.8 Preliminary data supports the use of topiramate, quetiapine, risperidone, ziprasidone, lamotrigine, and clonidine (Table 3).9-14 A recent review and meta-analysis showed efficacy with topira-mate, lamotrigine, valproate, aripiprazole, and olanzapine.15
For Ms. L, we restart venlafaxine at a lower dose of 50 mg/d and titrate it to 150 mg/d, which is still lower than her previous dose of 300 mg/d. She has no recurrence of seizures and her depression improves.
Table 2
Features of psychotherapeutic modalities for BPD
| Description | Mode of treatment | Skills taught | |
|---|---|---|---|
| Dialectical behavior therapy | Manualized, time-limited, cognitive-behavioral approach based on the biosocial theory of BPD | Individual therapy, group skills training, telephone contact, and therapist consultation | Core mindfulness skills, interpersonal effectiveness skills, emotion modulation skills, and distress tolerance skills |
| Systems Training for Emotional Predictability and Problem Solving | Manual-based, group treatment that includes a systems component to train family members, friends, and significant others | 20-week basic skills group and a 1-year, twice-monthly advanced group program; utilizes a classroom ‘seminar’ format | Awareness of illness, emotion management skills, and behavior management skills |
| BPD: borderline personality disorder | |||
Table 3
Pharmacotherapy for BPD: What the evidence says
| Study | Design | Results |
|---|---|---|
| Hollander et al, 20039 | 96 patients with Cluster B personality disorders randomized to divalproex or placebo for 12 weeks | Divalproex was superior to placebo in treating impulsive aggression, irritability, and global severity |
| Hilger et al, 200310 | Case report of 2 women with BPD and severe self-mutilation receiving quetiapine monotherapy | Quetiapine resulted in a marked improvement of impulsive behavior and overall level of function |
| Rizvi, 200211 | Case report of a 14-year-old female with borderline personality traits admitted to an inpatient facility for suicide attempt, impulsive behavior, and mood lability. Lamotrigine was started at 25 mg/d and titrated to 200 mg/d. At admission, she was receiving clonazepam, valproic acid, quetiapine, and fluoxetine, which were tapered and discontinued | Over 6 months of inpatient treatment, suicidal behavior and ideation diminished and impulse control and mood lability improved; continued improvement at 1-year follow up |
| Rocca et al, 200212 | 15 BPD outpatients with aggressive behavior given risperidone (mean dose 3.27 mg/d) in an 8-week open-label study | Risperidone produced a significant reduction in aggression based on AQ scores, reduction in depressive symptoms, and an increase in energy and global functioning |
| Philipsen et al, 200413 | 14 women with BPD given oral clonidine, 75 and 150 µg, while experiencing strong aversive inner tension and urge to commit self-injury | Clonidine significantly decreased aversive inner tension, dissociative symptoms, and urge to commit self-injury as measured by self rated scales |
| Pascual et al, 200414 | A 2-week open-label study of 10 females and 2 males presenting to psychiatric emergency service for self-injurious behavior, aggression/hostility, loss of impulse control, and severe anxiety/depressive symptoms received IM ziprasidone, 20 mg, followed by flexible oral dosing between 40 mg/d and 160 mg/d | 9 patients who completed the study showed statistically significant improvements on CGI-S, HAM-D-17, HAM-A, BPRS, and BIS |
| AQ: Aggression Questionnaire; BIS: Barratt Impulsiveness Scale; BPD: borderline personality disorder; BPRS: Brief Psychiatric Rating Scale; CGI-S: Clinical Global Impressions-Severity of Illness; HAM-A: Hamilton Anxiety Rating scale; HAM-D-17: 17-item Hamilton Depression Rating scale | ||
OUTCOME: Some improvement
Ms. L has no dramatic suicidal gestures for 3 years. Although she continues to engage in self-injurious behaviors, the intensity and frequency are reduced and she does not inflict any serious injury for 18 months. Her mood and behavior continue to oscillate; she is relatively calm and satisfied 1 week, angry and assaultive the next. This stormy course is expected given her BPD diagnosis.
Initially, Ms. L resided in a locked residential unit and was minimally compliant with treatment recommendations and unit policies. As treatment progressed she moved to a different locked unit and eventually to an apartment. Recently, she was placed in a more restrictive setting because her hostile and self-destructive behavior escalated.
The authors’ observations
Ms. L is no different from most Axis II Cluster B disordered patients. During treatment she shows improvement by refraining from self-destructive behaviors for up to 18 months, but she then briefly reverts back to maladaptive behaviors. Ms. L resides in a very structured treatment setting. It is not clear if the gains she made in treatment would have been possible if she was living on her own in the community.
One year after finishing the court-mandated “911 program,” Ms. L lives in the community, draws and paints quite well, attends weekly individual and group therapy, and refrains from self-mutilation. She still experiences volatile moods, but can handle them without inflicting self injury.
Related resources
- Oldham JM. Guideline watch: practice guideline for the treatment of patients with borderline personality disorder. Arlington, VA: American Psychiatric Association; 2005. www.psychiatryonline.com/content.aspx?aID=148722.
- Koenigsberg HW, Kernberg OF, Stone MH, et al. Borderline patients: extending the limits of treatability. New York, NY: Basic Books; 2000.
Drug brand names
- Aripiprazole • Abilify
- Buspirone • Buspar
- Carbamazepine • Tegretol
- Clonidine • Catapres
- Divalproex • Depakote
- Fluoxetine • Prozac
- Fluoxetine-olanzapine • Symbyax
- Lamotrigine • Lamictal
- Lithium • Eskalith, Lithobid
- Lorazepam • Ativan
- Olanzapine • Zyprexa
- Quetiapine • Seroquel
- Pregabalin • Lyrica
- Risperidone • Risperdal
- Topiramate • Topamax
- Valproic acid • Depakene
- Venlafaxine XR • Effexor XR
- Ziprasidone • Geodon
Disclosures
Dr. Hashmi is on the speakers bureau for AstraZeneca, Eli Lilly and Company, and Janssen.
Dr. Vowell reports no financial relationship with any company whose products are mentioned in this article or with manufacturers of competing products.
1. Diagnostic and statistical manual of mental disorders, 4th ed, text revision. Washington, DC: American Psychiatric Association; 2000.
2. Koenigsberg HW, Kernberg OF, Stone MH, et al. Borderline patients: extending the limits of treatability. New York, NY: Basic Books; 2000.
3. Nee C, Farman S. Female prisoners with borderline personality disorder: some promising treatment developments. Crim Behav Ment Health. 2005;15:2-16.
4. Oldham JM, Bender DS, Skodol AE, et al. Testing an APA practice guideline: symptom-targeted medication utilization for patients with borderline personality disorder. J Psychiatr Pract. 2004;10:156-161.
5. American Psychiatric Association Practice Guidelines. Practice guideline for the treatment of patients with borderline personality disorder. Am J Psychiatry. 2001;158(suppl 10):1-52.
6. Yatham LN. Newer anticonvulsants in the treatment of bipolar disorder. J Clin Psychiatry. 2004;65(suppl 10):28-35.
7. Rinne T, van den Brink W, Wouters L, et al. SSRI treatment of borderline personality disorder: a randomized, placebo-controlled clinical trial for female patients with borderline personality disorder. Am J Psychiatry. 2002;159(12):2048-2054.
8. Zanarini MC, Frankenburg FR, Parachini EA. A preliminary, randomized trial of fluoxetine, olanzapine, and the olanzapine-fluoxetine combination in women with borderline personality disorder. J Clin Psychiatry. 2004;65(7):903-907.
9. Hollander E, Tracy KA, Swann AC, et al. Divalproex in the treatment of impulsive aggression: efficacy in cluster B personality disorders. Neuropsychopharmacology. 2003;28(6):1186-1197.
10. Hilger E, Barnas C, Kasper S. Quetiapine in the treatment of borderline personality disorder. World J Biol Psychiatry. 2003;4(1):42-44.
11. Rizvi ST. Lamotrigine and borderline personality disorder. J Child Adolesc Psychopharmacol. 2002;12(4):365-366.
12. Rocca P, Marchiaro L, Cocuzza E, et al. Treatment of borderline personality disorder with risperidone. J Clin Psychiatry. 2002;63(3):241-244.
13. Philipsen A, Richter H, Schmahl C, et al. Clonidine in acute aversive inner tension and self-injurious behavior in female patients with borderline personality disorder. J Clin Psychiatry. 2004;65(10):1414-1419.
14. Pascual JC, Oller S, Soler J, et al. Ziprasidone in the acute treatment of borderline personality disorder in psychiatric emergency services. J Clin Psychiatry. 2004;65(9):1281-1282.
15. Lieb K, Völlm B, Rücker G, et al. Pharmacotherapy for borderline personality disorder: Cochrane systematic review of randomised trials. Br J Psychiatry. 2010;196(1):4-12.
CASE: Self-destructive behaviors
After being acquitted of 4 counts of second-degree forgery for writing checks from her mother’s bank account, Ms. L, age 52, is sent to the state hospital for a forensic examination to determine competency. Two years later she is granted conditional release from the hospital, transferred to our not-for-profit community mental health center, and enrolled in an intensive inpatient treatment program to monitor forensic patients. She is legally required to comply with treatment recommendations.
At admission, Ms. L is diagnosed with major depression, recurrent, and borderline personality disorder (BPD). She has no history of antisocial behavior or criminal acts other than forging checks and has never spent time in prison, which makes it unlikely she has co morbid antisocial personality disorder (Table 1).1
Over the next 5 years Ms. L tests limits with the treatment team and acts out by engaging in self-harming behaviors. In 1 instance, she cuts her forearm deeply, stuffs the wound with mayonnaise and paper towels, and wraps her arm with a bandage. She wears a long-sleeved shirt to hide her wound, which is not discovered until a severe infection develops.
Ms. L has difficulty with coping skills and interpersonal relationships. She approaches others with ambivalence and mistrust and consistently expects them to demean or take advantage of her. Ms. L is manipulative, at times injuring herself after perceived wrongdoings by staff. For example, after her therapist reschedules a meeting because of an emergency, Ms. L pours scalding water on her foot.
Table 1
Cluster B personality disorders: Differential diagnosis
| Diagnosis | Features |
|---|---|
| Borderline personality disorder | Self-destructiveness, angry disruptions in close relationships, and chronic feelings of deep emptiness and loneliness |
| Histrionic personality disorder | Attention seeking, manipulative behavior, and rapidly shifting emotions |
| Antisocial personality disorder | Manipulative to gain profit, power, or other material gratification |
| Source: Reference 1 | |
The authors’ observations
Ms. L consistently displays 3 common constructs of BPD:
- primitive defense mechanisms
- identity diffusion
- generally intact reality testing.2
Defense mechanisms are psychological attempts to deal with intrapsychic stress. Splitting—vacillating between extremes of idealization and devaluation—is a fundamental primitive defense mechanism that is the root of BPD.2 Identity diffusion causes confusion about life goals and values and feelings of boredom and emptiness. This internal world leads a patient to have the same perception of the external world, which explains many symptoms of BPD, such as rapidly shifting moods, intense anger, lack of clear sense of self, fear of abandonment, and unstable and intense interpersonal relationships.2
Early in treatment, Ms. L had difficulty breaking a cycle of self-defeating behavior, such as destroying personal items, trying to hang herself, and gluing an ear plug in her ear. During an argument with a staff member, Ms. L punched a wall and fractured her left hand. BPD patients sometimes will “up the ante” when acting out. For example, one of our patients claimed to have planted a bomb in an elementary school and another swallowed inedible objects, including spoons, forks, and butter knives. In Ms. L’s case, we addressed her self-harm behavior by helping her:
- develop less destructive coping skills such as drawing or painting
- identify irrational thoughts that contribute to self harm.
HISTORY: Troubled past
Raised by her biologic parents, Ms. L met all developmental milestones. She denies a history of childhood abuse but reports experiencing “depression and memory loss” and relationship problems with her parents during adolescence. As a child she often missed school because she “did not want anyone to know what a disgusting person I was” and “I should have my head cut open and cut into little pieces for thinking such mean thoughts.” Ms. L dropped out of school in the twelfth grade but obtained her general educational development certificate.
Notes and letters Ms. L wrote while in treatment consistently refer to her negative self-image. Ms. L writes that she feels she does not deserve to “be a part of this world,” is “never good enough for anyone,” and “should be thrown away with the garbage.”
Ms. L vacillates between desiring a closer relationship with her parents, especially her mother, and wanting to “cut them out of my life for good.” She has minimal contact with her older sister. Ms. L is divorced and has 2 adult sons. She was involved sporadically in her sons’ lives when they were children, but now has no contact with them.
BPD and crime
Ms. L is enrolled in the “911 program,” which monitors individuals who have been found not guilty by reason of mental defect. Individuals with BPD often are convicted of serious and violent crimes, which may be because of BPD features such as interpersonal hostility and self-harm. Impulsivity, substance abuse, and parental neglect—all of which are associated with BPD—can increase risk of criminality.3 There is no evidence to suggest a direct link between BPD and criminality; however, over-representation of BPD in prison populations suggest that in severe cases it may increase criminogenic risk.1,3
TREATMENT: Worsened depression
When Ms. L arrives at our facility, her medication regimen includes fluoxetine, 80 mg/d, risperidone, 2 mg/d, and buspirone, 20 mg/d. Risperidone and buspirone are discontinued because of perceived lack of efficacy. Venlafaxine XR is added and titrated to 300 mg/d, and Ms. L receives lorazepam, 1 and 2 mg as needed. However, lorazepam carries risks because impulsivity and impaired judgment—which are common in BPD—can lead to dependence and abuse. We feel that in a supervised setting the risks can be managed.
Recently, staff witnessed Ms. L experiencing an episode that appeared to be a grand mal seizure. After Ms. L is evaluated at the local emergency room, her EEG is normal, but a neurologic consult recommends discontinuing fluoxetine or venlafaxine XR because they may have contributed to the seizure. We taper and discontinue venlafaxine XR but Ms. L complains bitterly that she is getting increasingly depressed. On several occasions she attempts to pit team members against each other.
Ms. L falls, injures her back, and begins to abuse opiates. After her prescription runs out, she obtains more from an intellectually limited patient in her treatment program. Ms. L says she is getting more depressed, threatens suicide, and is placed in a more restrictive in-patient setting. We consider adding pregabalin to address her pain and help with anxiety and impulse control but the consulting neurologist prescribes carbamazepine, 400 mg/d, and her pain improves.5,6
The authors’ observations
BPD treatment primarily is psychotherapeutic and emphasizes skill building (Table 2) with focused, symptom-targeted pharmacotherapy as indicated.4 Pharmacotherapy typically targets 3 domains:
- affective dysregulation
- impulsive-behavioral dyscontrol symptoms
- cognitive-perceptual symptoms.
Patients with prominent anxiety may benefit from benzodiazepines, although research on these agents for BPD is limited. Recent studies show efficacy with fluoxetine, olanzapine, or a combination of both,7 and divalproex.8 Preliminary data supports the use of topiramate, quetiapine, risperidone, ziprasidone, lamotrigine, and clonidine (Table 3).9-14 A recent review and meta-analysis showed efficacy with topira-mate, lamotrigine, valproate, aripiprazole, and olanzapine.15
For Ms. L, we restart venlafaxine at a lower dose of 50 mg/d and titrate it to 150 mg/d, which is still lower than her previous dose of 300 mg/d. She has no recurrence of seizures and her depression improves.
Table 2
Features of psychotherapeutic modalities for BPD
| Description | Mode of treatment | Skills taught | |
|---|---|---|---|
| Dialectical behavior therapy | Manualized, time-limited, cognitive-behavioral approach based on the biosocial theory of BPD | Individual therapy, group skills training, telephone contact, and therapist consultation | Core mindfulness skills, interpersonal effectiveness skills, emotion modulation skills, and distress tolerance skills |
| Systems Training for Emotional Predictability and Problem Solving | Manual-based, group treatment that includes a systems component to train family members, friends, and significant others | 20-week basic skills group and a 1-year, twice-monthly advanced group program; utilizes a classroom ‘seminar’ format | Awareness of illness, emotion management skills, and behavior management skills |
| BPD: borderline personality disorder | |||
Table 3
Pharmacotherapy for BPD: What the evidence says
| Study | Design | Results |
|---|---|---|
| Hollander et al, 20039 | 96 patients with Cluster B personality disorders randomized to divalproex or placebo for 12 weeks | Divalproex was superior to placebo in treating impulsive aggression, irritability, and global severity |
| Hilger et al, 200310 | Case report of 2 women with BPD and severe self-mutilation receiving quetiapine monotherapy | Quetiapine resulted in a marked improvement of impulsive behavior and overall level of function |
| Rizvi, 200211 | Case report of a 14-year-old female with borderline personality traits admitted to an inpatient facility for suicide attempt, impulsive behavior, and mood lability. Lamotrigine was started at 25 mg/d and titrated to 200 mg/d. At admission, she was receiving clonazepam, valproic acid, quetiapine, and fluoxetine, which were tapered and discontinued | Over 6 months of inpatient treatment, suicidal behavior and ideation diminished and impulse control and mood lability improved; continued improvement at 1-year follow up |
| Rocca et al, 200212 | 15 BPD outpatients with aggressive behavior given risperidone (mean dose 3.27 mg/d) in an 8-week open-label study | Risperidone produced a significant reduction in aggression based on AQ scores, reduction in depressive symptoms, and an increase in energy and global functioning |
| Philipsen et al, 200413 | 14 women with BPD given oral clonidine, 75 and 150 µg, while experiencing strong aversive inner tension and urge to commit self-injury | Clonidine significantly decreased aversive inner tension, dissociative symptoms, and urge to commit self-injury as measured by self rated scales |
| Pascual et al, 200414 | A 2-week open-label study of 10 females and 2 males presenting to psychiatric emergency service for self-injurious behavior, aggression/hostility, loss of impulse control, and severe anxiety/depressive symptoms received IM ziprasidone, 20 mg, followed by flexible oral dosing between 40 mg/d and 160 mg/d | 9 patients who completed the study showed statistically significant improvements on CGI-S, HAM-D-17, HAM-A, BPRS, and BIS |
| AQ: Aggression Questionnaire; BIS: Barratt Impulsiveness Scale; BPD: borderline personality disorder; BPRS: Brief Psychiatric Rating Scale; CGI-S: Clinical Global Impressions-Severity of Illness; HAM-A: Hamilton Anxiety Rating scale; HAM-D-17: 17-item Hamilton Depression Rating scale | ||
OUTCOME: Some improvement
Ms. L has no dramatic suicidal gestures for 3 years. Although she continues to engage in self-injurious behaviors, the intensity and frequency are reduced and she does not inflict any serious injury for 18 months. Her mood and behavior continue to oscillate; she is relatively calm and satisfied 1 week, angry and assaultive the next. This stormy course is expected given her BPD diagnosis.
Initially, Ms. L resided in a locked residential unit and was minimally compliant with treatment recommendations and unit policies. As treatment progressed she moved to a different locked unit and eventually to an apartment. Recently, she was placed in a more restrictive setting because her hostile and self-destructive behavior escalated.
The authors’ observations
Ms. L is no different from most Axis II Cluster B disordered patients. During treatment she shows improvement by refraining from self-destructive behaviors for up to 18 months, but she then briefly reverts back to maladaptive behaviors. Ms. L resides in a very structured treatment setting. It is not clear if the gains she made in treatment would have been possible if she was living on her own in the community.
One year after finishing the court-mandated “911 program,” Ms. L lives in the community, draws and paints quite well, attends weekly individual and group therapy, and refrains from self-mutilation. She still experiences volatile moods, but can handle them without inflicting self injury.
Related resources
- Oldham JM. Guideline watch: practice guideline for the treatment of patients with borderline personality disorder. Arlington, VA: American Psychiatric Association; 2005. www.psychiatryonline.com/content.aspx?aID=148722.
- Koenigsberg HW, Kernberg OF, Stone MH, et al. Borderline patients: extending the limits of treatability. New York, NY: Basic Books; 2000.
Drug brand names
- Aripiprazole • Abilify
- Buspirone • Buspar
- Carbamazepine • Tegretol
- Clonidine • Catapres
- Divalproex • Depakote
- Fluoxetine • Prozac
- Fluoxetine-olanzapine • Symbyax
- Lamotrigine • Lamictal
- Lithium • Eskalith, Lithobid
- Lorazepam • Ativan
- Olanzapine • Zyprexa
- Quetiapine • Seroquel
- Pregabalin • Lyrica
- Risperidone • Risperdal
- Topiramate • Topamax
- Valproic acid • Depakene
- Venlafaxine XR • Effexor XR
- Ziprasidone • Geodon
Disclosures
Dr. Hashmi is on the speakers bureau for AstraZeneca, Eli Lilly and Company, and Janssen.
Dr. Vowell reports no financial relationship with any company whose products are mentioned in this article or with manufacturers of competing products.
CASE: Self-destructive behaviors
After being acquitted of 4 counts of second-degree forgery for writing checks from her mother’s bank account, Ms. L, age 52, is sent to the state hospital for a forensic examination to determine competency. Two years later she is granted conditional release from the hospital, transferred to our not-for-profit community mental health center, and enrolled in an intensive inpatient treatment program to monitor forensic patients. She is legally required to comply with treatment recommendations.
At admission, Ms. L is diagnosed with major depression, recurrent, and borderline personality disorder (BPD). She has no history of antisocial behavior or criminal acts other than forging checks and has never spent time in prison, which makes it unlikely she has co morbid antisocial personality disorder (Table 1).1
Over the next 5 years Ms. L tests limits with the treatment team and acts out by engaging in self-harming behaviors. In 1 instance, she cuts her forearm deeply, stuffs the wound with mayonnaise and paper towels, and wraps her arm with a bandage. She wears a long-sleeved shirt to hide her wound, which is not discovered until a severe infection develops.
Ms. L has difficulty with coping skills and interpersonal relationships. She approaches others with ambivalence and mistrust and consistently expects them to demean or take advantage of her. Ms. L is manipulative, at times injuring herself after perceived wrongdoings by staff. For example, after her therapist reschedules a meeting because of an emergency, Ms. L pours scalding water on her foot.
Table 1
Cluster B personality disorders: Differential diagnosis
| Diagnosis | Features |
|---|---|
| Borderline personality disorder | Self-destructiveness, angry disruptions in close relationships, and chronic feelings of deep emptiness and loneliness |
| Histrionic personality disorder | Attention seeking, manipulative behavior, and rapidly shifting emotions |
| Antisocial personality disorder | Manipulative to gain profit, power, or other material gratification |
| Source: Reference 1 | |
The authors’ observations
Ms. L consistently displays 3 common constructs of BPD:
- primitive defense mechanisms
- identity diffusion
- generally intact reality testing.2
Defense mechanisms are psychological attempts to deal with intrapsychic stress. Splitting—vacillating between extremes of idealization and devaluation—is a fundamental primitive defense mechanism that is the root of BPD.2 Identity diffusion causes confusion about life goals and values and feelings of boredom and emptiness. This internal world leads a patient to have the same perception of the external world, which explains many symptoms of BPD, such as rapidly shifting moods, intense anger, lack of clear sense of self, fear of abandonment, and unstable and intense interpersonal relationships.2
Early in treatment, Ms. L had difficulty breaking a cycle of self-defeating behavior, such as destroying personal items, trying to hang herself, and gluing an ear plug in her ear. During an argument with a staff member, Ms. L punched a wall and fractured her left hand. BPD patients sometimes will “up the ante” when acting out. For example, one of our patients claimed to have planted a bomb in an elementary school and another swallowed inedible objects, including spoons, forks, and butter knives. In Ms. L’s case, we addressed her self-harm behavior by helping her:
- develop less destructive coping skills such as drawing or painting
- identify irrational thoughts that contribute to self harm.
HISTORY: Troubled past
Raised by her biologic parents, Ms. L met all developmental milestones. She denies a history of childhood abuse but reports experiencing “depression and memory loss” and relationship problems with her parents during adolescence. As a child she often missed school because she “did not want anyone to know what a disgusting person I was” and “I should have my head cut open and cut into little pieces for thinking such mean thoughts.” Ms. L dropped out of school in the twelfth grade but obtained her general educational development certificate.
Notes and letters Ms. L wrote while in treatment consistently refer to her negative self-image. Ms. L writes that she feels she does not deserve to “be a part of this world,” is “never good enough for anyone,” and “should be thrown away with the garbage.”
Ms. L vacillates between desiring a closer relationship with her parents, especially her mother, and wanting to “cut them out of my life for good.” She has minimal contact with her older sister. Ms. L is divorced and has 2 adult sons. She was involved sporadically in her sons’ lives when they were children, but now has no contact with them.
BPD and crime
Ms. L is enrolled in the “911 program,” which monitors individuals who have been found not guilty by reason of mental defect. Individuals with BPD often are convicted of serious and violent crimes, which may be because of BPD features such as interpersonal hostility and self-harm. Impulsivity, substance abuse, and parental neglect—all of which are associated with BPD—can increase risk of criminality.3 There is no evidence to suggest a direct link between BPD and criminality; however, over-representation of BPD in prison populations suggest that in severe cases it may increase criminogenic risk.1,3
TREATMENT: Worsened depression
When Ms. L arrives at our facility, her medication regimen includes fluoxetine, 80 mg/d, risperidone, 2 mg/d, and buspirone, 20 mg/d. Risperidone and buspirone are discontinued because of perceived lack of efficacy. Venlafaxine XR is added and titrated to 300 mg/d, and Ms. L receives lorazepam, 1 and 2 mg as needed. However, lorazepam carries risks because impulsivity and impaired judgment—which are common in BPD—can lead to dependence and abuse. We feel that in a supervised setting the risks can be managed.
Recently, staff witnessed Ms. L experiencing an episode that appeared to be a grand mal seizure. After Ms. L is evaluated at the local emergency room, her EEG is normal, but a neurologic consult recommends discontinuing fluoxetine or venlafaxine XR because they may have contributed to the seizure. We taper and discontinue venlafaxine XR but Ms. L complains bitterly that she is getting increasingly depressed. On several occasions she attempts to pit team members against each other.
Ms. L falls, injures her back, and begins to abuse opiates. After her prescription runs out, she obtains more from an intellectually limited patient in her treatment program. Ms. L says she is getting more depressed, threatens suicide, and is placed in a more restrictive in-patient setting. We consider adding pregabalin to address her pain and help with anxiety and impulse control but the consulting neurologist prescribes carbamazepine, 400 mg/d, and her pain improves.5,6
The authors’ observations
BPD treatment primarily is psychotherapeutic and emphasizes skill building (Table 2) with focused, symptom-targeted pharmacotherapy as indicated.4 Pharmacotherapy typically targets 3 domains:
- affective dysregulation
- impulsive-behavioral dyscontrol symptoms
- cognitive-perceptual symptoms.
Patients with prominent anxiety may benefit from benzodiazepines, although research on these agents for BPD is limited. Recent studies show efficacy with fluoxetine, olanzapine, or a combination of both,7 and divalproex.8 Preliminary data supports the use of topiramate, quetiapine, risperidone, ziprasidone, lamotrigine, and clonidine (Table 3).9-14 A recent review and meta-analysis showed efficacy with topira-mate, lamotrigine, valproate, aripiprazole, and olanzapine.15
For Ms. L, we restart venlafaxine at a lower dose of 50 mg/d and titrate it to 150 mg/d, which is still lower than her previous dose of 300 mg/d. She has no recurrence of seizures and her depression improves.
Table 2
Features of psychotherapeutic modalities for BPD
| Description | Mode of treatment | Skills taught | |
|---|---|---|---|
| Dialectical behavior therapy | Manualized, time-limited, cognitive-behavioral approach based on the biosocial theory of BPD | Individual therapy, group skills training, telephone contact, and therapist consultation | Core mindfulness skills, interpersonal effectiveness skills, emotion modulation skills, and distress tolerance skills |
| Systems Training for Emotional Predictability and Problem Solving | Manual-based, group treatment that includes a systems component to train family members, friends, and significant others | 20-week basic skills group and a 1-year, twice-monthly advanced group program; utilizes a classroom ‘seminar’ format | Awareness of illness, emotion management skills, and behavior management skills |
| BPD: borderline personality disorder | |||
Table 3
Pharmacotherapy for BPD: What the evidence says
| Study | Design | Results |
|---|---|---|
| Hollander et al, 20039 | 96 patients with Cluster B personality disorders randomized to divalproex or placebo for 12 weeks | Divalproex was superior to placebo in treating impulsive aggression, irritability, and global severity |
| Hilger et al, 200310 | Case report of 2 women with BPD and severe self-mutilation receiving quetiapine monotherapy | Quetiapine resulted in a marked improvement of impulsive behavior and overall level of function |
| Rizvi, 200211 | Case report of a 14-year-old female with borderline personality traits admitted to an inpatient facility for suicide attempt, impulsive behavior, and mood lability. Lamotrigine was started at 25 mg/d and titrated to 200 mg/d. At admission, she was receiving clonazepam, valproic acid, quetiapine, and fluoxetine, which were tapered and discontinued | Over 6 months of inpatient treatment, suicidal behavior and ideation diminished and impulse control and mood lability improved; continued improvement at 1-year follow up |
| Rocca et al, 200212 | 15 BPD outpatients with aggressive behavior given risperidone (mean dose 3.27 mg/d) in an 8-week open-label study | Risperidone produced a significant reduction in aggression based on AQ scores, reduction in depressive symptoms, and an increase in energy and global functioning |
| Philipsen et al, 200413 | 14 women with BPD given oral clonidine, 75 and 150 µg, while experiencing strong aversive inner tension and urge to commit self-injury | Clonidine significantly decreased aversive inner tension, dissociative symptoms, and urge to commit self-injury as measured by self rated scales |
| Pascual et al, 200414 | A 2-week open-label study of 10 females and 2 males presenting to psychiatric emergency service for self-injurious behavior, aggression/hostility, loss of impulse control, and severe anxiety/depressive symptoms received IM ziprasidone, 20 mg, followed by flexible oral dosing between 40 mg/d and 160 mg/d | 9 patients who completed the study showed statistically significant improvements on CGI-S, HAM-D-17, HAM-A, BPRS, and BIS |
| AQ: Aggression Questionnaire; BIS: Barratt Impulsiveness Scale; BPD: borderline personality disorder; BPRS: Brief Psychiatric Rating Scale; CGI-S: Clinical Global Impressions-Severity of Illness; HAM-A: Hamilton Anxiety Rating scale; HAM-D-17: 17-item Hamilton Depression Rating scale | ||
OUTCOME: Some improvement
Ms. L has no dramatic suicidal gestures for 3 years. Although she continues to engage in self-injurious behaviors, the intensity and frequency are reduced and she does not inflict any serious injury for 18 months. Her mood and behavior continue to oscillate; she is relatively calm and satisfied 1 week, angry and assaultive the next. This stormy course is expected given her BPD diagnosis.
Initially, Ms. L resided in a locked residential unit and was minimally compliant with treatment recommendations and unit policies. As treatment progressed she moved to a different locked unit and eventually to an apartment. Recently, she was placed in a more restrictive setting because her hostile and self-destructive behavior escalated.
The authors’ observations
Ms. L is no different from most Axis II Cluster B disordered patients. During treatment she shows improvement by refraining from self-destructive behaviors for up to 18 months, but she then briefly reverts back to maladaptive behaviors. Ms. L resides in a very structured treatment setting. It is not clear if the gains she made in treatment would have been possible if she was living on her own in the community.
One year after finishing the court-mandated “911 program,” Ms. L lives in the community, draws and paints quite well, attends weekly individual and group therapy, and refrains from self-mutilation. She still experiences volatile moods, but can handle them without inflicting self injury.
Related resources
- Oldham JM. Guideline watch: practice guideline for the treatment of patients with borderline personality disorder. Arlington, VA: American Psychiatric Association; 2005. www.psychiatryonline.com/content.aspx?aID=148722.
- Koenigsberg HW, Kernberg OF, Stone MH, et al. Borderline patients: extending the limits of treatability. New York, NY: Basic Books; 2000.
Drug brand names
- Aripiprazole • Abilify
- Buspirone • Buspar
- Carbamazepine • Tegretol
- Clonidine • Catapres
- Divalproex • Depakote
- Fluoxetine • Prozac
- Fluoxetine-olanzapine • Symbyax
- Lamotrigine • Lamictal
- Lithium • Eskalith, Lithobid
- Lorazepam • Ativan
- Olanzapine • Zyprexa
- Quetiapine • Seroquel
- Pregabalin • Lyrica
- Risperidone • Risperdal
- Topiramate • Topamax
- Valproic acid • Depakene
- Venlafaxine XR • Effexor XR
- Ziprasidone • Geodon
Disclosures
Dr. Hashmi is on the speakers bureau for AstraZeneca, Eli Lilly and Company, and Janssen.
Dr. Vowell reports no financial relationship with any company whose products are mentioned in this article or with manufacturers of competing products.
1. Diagnostic and statistical manual of mental disorders, 4th ed, text revision. Washington, DC: American Psychiatric Association; 2000.
2. Koenigsberg HW, Kernberg OF, Stone MH, et al. Borderline patients: extending the limits of treatability. New York, NY: Basic Books; 2000.
3. Nee C, Farman S. Female prisoners with borderline personality disorder: some promising treatment developments. Crim Behav Ment Health. 2005;15:2-16.
4. Oldham JM, Bender DS, Skodol AE, et al. Testing an APA practice guideline: symptom-targeted medication utilization for patients with borderline personality disorder. J Psychiatr Pract. 2004;10:156-161.
5. American Psychiatric Association Practice Guidelines. Practice guideline for the treatment of patients with borderline personality disorder. Am J Psychiatry. 2001;158(suppl 10):1-52.
6. Yatham LN. Newer anticonvulsants in the treatment of bipolar disorder. J Clin Psychiatry. 2004;65(suppl 10):28-35.
7. Rinne T, van den Brink W, Wouters L, et al. SSRI treatment of borderline personality disorder: a randomized, placebo-controlled clinical trial for female patients with borderline personality disorder. Am J Psychiatry. 2002;159(12):2048-2054.
8. Zanarini MC, Frankenburg FR, Parachini EA. A preliminary, randomized trial of fluoxetine, olanzapine, and the olanzapine-fluoxetine combination in women with borderline personality disorder. J Clin Psychiatry. 2004;65(7):903-907.
9. Hollander E, Tracy KA, Swann AC, et al. Divalproex in the treatment of impulsive aggression: efficacy in cluster B personality disorders. Neuropsychopharmacology. 2003;28(6):1186-1197.
10. Hilger E, Barnas C, Kasper S. Quetiapine in the treatment of borderline personality disorder. World J Biol Psychiatry. 2003;4(1):42-44.
11. Rizvi ST. Lamotrigine and borderline personality disorder. J Child Adolesc Psychopharmacol. 2002;12(4):365-366.
12. Rocca P, Marchiaro L, Cocuzza E, et al. Treatment of borderline personality disorder with risperidone. J Clin Psychiatry. 2002;63(3):241-244.
13. Philipsen A, Richter H, Schmahl C, et al. Clonidine in acute aversive inner tension and self-injurious behavior in female patients with borderline personality disorder. J Clin Psychiatry. 2004;65(10):1414-1419.
14. Pascual JC, Oller S, Soler J, et al. Ziprasidone in the acute treatment of borderline personality disorder in psychiatric emergency services. J Clin Psychiatry. 2004;65(9):1281-1282.
15. Lieb K, Völlm B, Rücker G, et al. Pharmacotherapy for borderline personality disorder: Cochrane systematic review of randomised trials. Br J Psychiatry. 2010;196(1):4-12.
1. Diagnostic and statistical manual of mental disorders, 4th ed, text revision. Washington, DC: American Psychiatric Association; 2000.
2. Koenigsberg HW, Kernberg OF, Stone MH, et al. Borderline patients: extending the limits of treatability. New York, NY: Basic Books; 2000.
3. Nee C, Farman S. Female prisoners with borderline personality disorder: some promising treatment developments. Crim Behav Ment Health. 2005;15:2-16.
4. Oldham JM, Bender DS, Skodol AE, et al. Testing an APA practice guideline: symptom-targeted medication utilization for patients with borderline personality disorder. J Psychiatr Pract. 2004;10:156-161.
5. American Psychiatric Association Practice Guidelines. Practice guideline for the treatment of patients with borderline personality disorder. Am J Psychiatry. 2001;158(suppl 10):1-52.
6. Yatham LN. Newer anticonvulsants in the treatment of bipolar disorder. J Clin Psychiatry. 2004;65(suppl 10):28-35.
7. Rinne T, van den Brink W, Wouters L, et al. SSRI treatment of borderline personality disorder: a randomized, placebo-controlled clinical trial for female patients with borderline personality disorder. Am J Psychiatry. 2002;159(12):2048-2054.
8. Zanarini MC, Frankenburg FR, Parachini EA. A preliminary, randomized trial of fluoxetine, olanzapine, and the olanzapine-fluoxetine combination in women with borderline personality disorder. J Clin Psychiatry. 2004;65(7):903-907.
9. Hollander E, Tracy KA, Swann AC, et al. Divalproex in the treatment of impulsive aggression: efficacy in cluster B personality disorders. Neuropsychopharmacology. 2003;28(6):1186-1197.
10. Hilger E, Barnas C, Kasper S. Quetiapine in the treatment of borderline personality disorder. World J Biol Psychiatry. 2003;4(1):42-44.
11. Rizvi ST. Lamotrigine and borderline personality disorder. J Child Adolesc Psychopharmacol. 2002;12(4):365-366.
12. Rocca P, Marchiaro L, Cocuzza E, et al. Treatment of borderline personality disorder with risperidone. J Clin Psychiatry. 2002;63(3):241-244.
13. Philipsen A, Richter H, Schmahl C, et al. Clonidine in acute aversive inner tension and self-injurious behavior in female patients with borderline personality disorder. J Clin Psychiatry. 2004;65(10):1414-1419.
14. Pascual JC, Oller S, Soler J, et al. Ziprasidone in the acute treatment of borderline personality disorder in psychiatric emergency services. J Clin Psychiatry. 2004;65(9):1281-1282.
15. Lieb K, Völlm B, Rücker G, et al. Pharmacotherapy for borderline personality disorder: Cochrane systematic review of randomised trials. Br J Psychiatry. 2010;196(1):4-12.
Impaired physicians: How to recognize, when to report, and where to refer
From all outward appearances Dr. S, a part-time psychiatrist at an inpatient psychiatric facility and in private practice for 12 years, is living the “perfect life,” with a wife, children, and successful practice.
In retrospect, his drug addiction began insidiously. In college, Dr. S continued to use oxycodone/acetaminophen prescribed for a shoulder injury long after his pain had resolved. He began to use cocaine in residency to help him “get through” the 36-hour call days, and it “helped” him earn the chief resident position because of his heightened energy and concentration. Dr. S’ primary care physician initially prescribed him benzodiazepines for anxiety and to help him sleep. Opiates were prescribed for a musculoskeletal injury. Dr. S obtained prescriptions for these medications from multiple providers. This ultimately escalated to self-prescription using aliases. Dr. S also began to drink heavily each evening.
Dr. S disregards colleagues’ comment about his obvious mood swings, which he attributes to his stressful job and “nagging” wife, despite having a family history of bipolar disorder. He becomes enraged when his wife or friends suggest he seek help. His colleagues whisper behind his back, but for years, no one confronts him about his unpredictable and frequently inappropriate behavior. Eventually, a nurse files a sexual harassment suit against Dr. S, and a patient complains to the medical board that Dr. S exhibited sexually inappropriate behavior during a therapy session.
As physicians, recognizing impairment in our colleagues or ourselves can be difficult. The American Medical Association defines an impaired physician as one who is unable to fulfill personal or professional responsibilities because of psychiatric illness, alcoholism, or drug dependence.1 Impairment is present when a physician is unable to perform in a manner that conforms to acceptable standards of practice, exhibits serious flaws in judgment, and provides incompetent care.1-3
Recognizing when a physician is impaired, deciding whether to report him or her to the state medical board, and referring a colleague for treatment can be challenging. This article will:
- review substance abuse, cognitive decline, and other causes of impairment
- address legal and ethical issues involved in reporting a colleague to the state medical board
- provide resources for physician treatment and assistance.
Physicians and addiction
Chemical dependence is the most frequent disabling illness among physicians,4 and substance abuse is the most common form of impairment that results in discipline by a state medical board.5 An estimated 6% to 8% of physicians abuse drugs, and approximately 14% develop alcohol use disorders; these rates are comparable to those of the general population.5 Psychiatrists, emergency room physicians, and solo practitioners are 3 times more likely to abuse substances than other doctors.6 An obsessive-compulsive personality and other factors may predispose physicians to substance abuse (Table 1).7,8
Alcohol is the most commonly abused substance, followed by opiates, cocaine, and other stimulants.9 Physicians are estimated to use opiates and benzodiazepines at a rate 5 times greater than that of the general public.10,11
An often-hidden problem. Physicians frequently deny substance abuse and many are able to conceal the problem from coworkers, even as their personal lives disintegrate.1,12 Marital and relationship problems may be the first indication of impairment, which gradually spreads to other aspects of their lives (Table 2).1,2,5 A doctor’s professional performance often is the last area to be affected.1,12
Substance abuse in physicians may long go unreported. The clinician’s family may want to protect the physician’s reputation, career, and income. Colleagues may be intimidated, uncertain of their concerns, or fearful for their jobs if they report the physician’s impairment. Patients may be reluctant to report their concerns because they depend on their provider for health care, respect the physician, or deny that a doctor could have a drug or alcohol problem.5
Table 1
Physicians and substance abuse: Predisposing factors
| Obsessive-compulsive personality style |
| Family history of substance use disorders or mental illness |
| Childhood family problems |
| Personal mental illness |
| Sensation-seeking behavior |
| Denial of personal and social problems |
| Perfectionism |
| Idealism |
| Source: References 7,8 |
Table 2
Signs of substance abuse
| Frequent tardiness and absences |
| Unexplained disappearances during working hours |
| Inappropriate behavior |
| Affective lability or irritability |
| Interpersonal conflict |
| Avoidance of peers or supervisors |
| Keeping odd hours |
| Disorganization and forgetfulness |
| Diminished chart completion and work performance |
| Heavy drinking at social functions |
| Unexplained changes in weight or energy level |
| Diminished personal hygiene |
| Slurred or rapid speech |
| Frequently dilated pupils or red and watery eyes and a runny nose |
| Defensiveness, anxiety, apathy, or manipulative behavior |
| Withdrawal from long-standing relationships |
| Source: References 1,2,5 |
Screening for cognitive decline
Many people with cognitive impairment lack insight into their problem and may minimize or deny the degree of their impairment.13 The prevalence of dementia in individuals age ≥65 is 3% to 11%,14 and 18% of physicians are in this age group.15
Ethical, legal, and practical issues arise in determining who, when, and how to screen physicians for cognitive problems. Standard screening exams may not be adequately sensitive for a well-educated physician, and neuropsychological testing may be necessary to detect mild cognitive impairment.13 In addition, the cognitive, visual-spacial, reactivity, reasoning, and calculation skills required for capable medical practice vary among specialties.16
One screening option is a “360-degree review” of information obtained from peers, patients, and non-physician colleagues that the College of Physicians and Surgeons in Alberta, Canada, has incorporated into its Physician’s Achievement Review (PAR) for physicians age ≥65.17 Compiled in a confidential manner and shared with the physician, the 360-degree survey assesses his or her:
- skill and knowledge
- psychosocial functioning
- management skills
- performance
- collegiality.
In Alberta, physicians who score in the lower one-third of the survey are assessed with an on-site evaluation by physicians from his or her specialty appointed by the PAR Director of Practice Improvement.
Alternately, physicians age ≥65 could be required to undergo annual or biannual neuropsychological testing to screen for mild cognitive impairment or other evidence of cognitive decline. In the United States, any screening requirement must be structured to comply with the Age Discrimination in Employment Act.18
If a physician shows evidence of cognitive impairment, the state medical board should initiate closer scrutiny and modify or revoke privileges if indicated. Remediation programs designed to assist impaired physicians may not be effective for those with cognitive impairment because the decline in cognitive functioning associated with illnesses such as Alzheimer’s disease often is progressive.13
‘Disruptive’ physicians
Mental illnesses such as personality or affective disorders, interpersonal problems within or outside the workplace, or other stressors could lead a physician to disrupt the workplace or patient care. Numerous programs have been established across the United States to help evaluate and treat disruptive physicians. Remediation programs can help identify and offer education for “dyscompetent” physicians (see Related Resources).13
To report, or not to report
In a national survey of physicians, only 45% of respondents indicated that they had notified the state licensing board of a colleague they felt was impaired or incompetent, yet almost all (96%) indicated that these individuals should be reported.19 Any duty to report requires, at minimum, that the physician be affected by an illness that impairs his or her cognition, concentration, rapid decision making, and ability to handle emergencies or perform work functions safely.20
Shouten20 cautions that someone who is considering filing a report because of fear of liability if they don’t should balance this concern against potential liability for breaching confidentiality. If there is evidence of an imminent risk or serious harm to the physician or patients, you may be legally required to breach confidentiality. Some states require licensed health practitioners to report acts of professional misconduct, unless the information is obtained solely from directly treating the physician. These requirements apply only within the state, and only to that state’s licensees.20-22
An ethical requirement to report also must be balanced against the obligation to maintain confidentiality. Ethics are largely a matter of individual standards, and individuals’ perceived ethical duties vary.
If you are considering reporting an impaired colleague, learn the laws in your state. If the physician is from another jurisdiction, the law provides little definitive guidance. Shouten recommends focusing on clinical outcomes for the doctor and his or her patients rather than on legal liability.20
Physician health programs
Nationwide directories of physician health programs are available from the Federation of State Physician Health Programs and the Federation of State Medical Boards (see Related Resources). Some programs are affiliated with state licensing boards, some are branches of state medical societies, and some are independent. These programs provide confidential treatment and assistance to practitioners experiencing substance or alcohol abuse, mental illness, or disruptive behavior. Some institutions may offer physicians an employee assistance program.
State medical societies may provide information about accessing a physician health program. Programs sponsored by medical societies almost always are independent of state licensing boards. This arrangement allows physicians to seek help without fear of punishment or censure or revocation of their license. Noncompliance with a physician health program, however, likely will result in being reported to the medical board.
Physician health programs typically employ a rehabilitative approach. Punitive measures such as reporting physicians to the medical board usually are not pursued unless the individual does not comply with treatment and monitoring guidelines. A physician who abuses substances, for example, may be required to complete a residential treatment program, attend support group meetings such as 12-step programs, participate in individual therapy, and undergo random screening for alcohol and illicit drug use.5
Abstinence is the goal of treating clinicians who abuse substances. Physicians have better outcomes than the general population, with reported abstinence rates of 70% to 90% for those who complete treatment;23,24 75% to 85% of physicians who complete rehabilitation and comply with close monitoring and follow-up care are able to return to work.24,25 Acceptance of recovery as a lifelong process, monitoring, and self-vigilance often are necessary to achieve and maintain abstinence.5
Risk factors for relapse include:
- denial of illness
- poor stress-coping and relationship skills
- social and professional isolation
- inability to accept feedback
- complacency and overconfidence
- failure to attend support group meetings
- dysfunctional family dynamics
- feelings of self-pity, blame, and guilt.5
Treating an impaired colleague. Reid26 recommends that psychiatrists should not evaluate or treat a self-referred, potentially impaired physician unless the relationship is strictly clinical. A physician may withhold symptoms, behaviors, or problems because his or her license, malpractice case, or career are at stake.
Advise a physician who requests evaluation or treatment related to license concerns or any legal matter to seek legal counsel. Working with such physician/patients only upon referral by a lawyer, licensing board, or physicians’ health committee provides treating psychiatrists with a clear professional role, allowing them to focus solely on the physician/patient’s treatment needs.
CASE CONTINUED: Extensive help, then success
The medical director of the hospital where Dr. S works refers him to his state’s impaired physician program. After investigating the complaints by the nurse and patient, the medical board suspends Dr. S’ license and requires him to enter a substance abuse treatment program. He completes an intensive residential program for impaired physicians and achieves sobriety from drugs and alcohol. His mood disorder is successfully treated with medications and psychotherapy. The medical board requires Dr. S to have a chaperone present for all visits with patients and submit random urine drug screens once his license is provisionally restored. The medical board also requires Dr. S to undergo ongoing psychiatric care and medication monitoring. He remains abstinent from alcohol and drugs, complies with the medical board’s requirements, and enjoys a productive practice and improved relationship with his family.
Disruptive physicians
- Samenow C, Swiggart W, Spickard A Jr. Consequence of physician disruptive behavior. Tenn Med. 2007;100(11):38-40.
- Leape LL, Fromson JA. Problem doctors: is there a system-level solution? Ann Intern Med. 2006;144(2):107-115.
- Linney BJ. Confronting the disruptive physician. Physician Exec. 1997;23:55-59.
Physician evaluation
- Anfang SA, Faulkner LR, Fromson JA, et al. The American Psychiatric Association’s resource document on guidelines for psychiatric fitness-for-duty evaluations of physicians. J Am Acad Psychiatry Law. 2005;33:85-88.
- Harmon L, Pomm R. Evaluation, treatment, and monitoring of disruptive physicians’ behavior. Psychiatr Ann. 2004; 34:770-774.
Other resources
- Federation of State Physician Health Programs. www.fsphp.org.
- Federation of State Medical Boards: Directory of Physician Assessment and Remedial Education Programs. www.fsmb.org/pdf/RemEdProg.pdf.
- The Center for Professional Health, Vanderbilt University Medical Center. www.mc.vanderbilt.edu/cph.
- Vanderbilt Comprehensive Assessment Program. www.mc.vanderbilt.edu/root/vcap.
- Alcoholics Anonymous. www.aa.org.
- Narcotics Anonymous. www.na.org.
Drug Brand Name
- Oxycodone/acetaminophen • Percocet
Disclosure
The authors report no financial relationship with the manufacturer of any product mentioned in this article or with manufacturers of competing products.
1. Breiner SJ. The impaired physician. J Med Educ. 1979;54:673.-
2. Talbott GD, Gallegos KV, Angres DH. Impairment and recovery in physicians and other health professionals. In: Graham AW, Schultz TK, eds. Principles of addiction medicine. 2nd ed. Chevy Chase, MD: American Society of Addiction Medicine, Inc; 1998:1263-1277.
3. American Medical Association Council on Mental Health. The sick physician: impairment by psychiatric disorders, including alcoholism and drug dependence. JAMA. 1973;223:684-687.
4. Talbott G, Wright C. Chemical dependency in healthcare professionals. Occup Med. 1987;2:581-591.
5. Baldisseri MR. Impaired healthcare professional. Crit Care Med. 2007;35:S106-116.
6. Mansky PA. Physician health programs and the potentially impaired physician with a substance use disorder. Psychiatr Serv. 1996;47:465-467.
7. Boisaubin EV, Levine RE. Identifying and assisting the impaired physician. Am J Med Sci. 2001;322:31-36.
8. Bissel L, Jones RW. The alcoholic physician: a survey. Am J Psychiatry. 1976;133:1142-1146.
9. Robins L, Reiger D. Psychiatric disorders in America: the Epidemiologic Catchment Area Study. New York, NY: The Free Press; 1991.
10. Gallegos KV, Browne CH, Veit FW, et al. Addiction in anesthesiologists: drug access and patterns of substance abuse. QRB Qual Rev Bull. 1988;14:116-122.
11. Hughes PH, Brandenburg N, Baldwin DC, et al. Prevalence of substance abuse among U.S. physicians. JAMA. 1992;267:2333-2339.
12. Vaillant GE, Clark W, Cyrus C, et al. Prospective study of alcoholism treatment. Eight year follow-up. Am J Med. 1983;75:455-463.
13. LoboPrabhu SM, Molinari VA, Hamilton JD, et al. The aging physician with cognitive impairment: approaches to oversight, prevention and remediation. Am J Geriatr Psychiatry. 2009;17:445-454.
14. U.S. General Accounting Office. Alzheimer’s disease: estimates of prevalence in the United States. Washington, DC: U.S. General Accounting Office; 1998:98. Publication GAO/HEHS-98-16.
15. American Medical Association. Physician characteristics and distribution in the U.S., 2006. Washington, DC: American Medical Association; 2006.
16. Blasier R. The problem of the aging surgeon. Clin Orthop Relat Res. 2009;467:402-411.
17. College of Physicians and Surgeons of Alberta. Physician Achievement Review (PAR) Program. Available at: http://www.cpsa.ab.ca/Services/PARprogram/Overview.aspx. Accessed March 27, 2010.
18. The Age Discrimination in Employment Act. (Vol. Pub. L. No. 90-202, 81 Stat. 602 (Dec. 15, 1967), codified as Chapter 14 of Title 29 of the United States Code, 29 U.S.C. § 621 through 29 U.S.C. § 63), 1967.
19. Campbell EG, Regan S, Gruen RL, et al. Professionalism in medicine: results of a national survey of physicians. Ann Int Med. 2007;147:795-802.
20. Shouten R. Impaired physicians: is there a duty to report to state licensing boards? Harvard Rev Psychiatry. 2000;8:36-39.
21. Mass Gen Laws ch 112 § 5F.
22. NY PHL § 230 (11) (e).
23. Femino J, Nirenberg TD. Treatment outcome studies on physician impairment: a review of the literature. R I Med. 1994;77:345-350.
24. Alpern F, Correnti CE, Dolan TE, et al. A survey of recovering Maryland physicians. Md Med J. 1992;41:301-303.
25. Gallegos KV, Lubin BH, Bowers C, et al. Relapse and recovery: five to ten year follow-up study of chemically dependent physicians—the Georgia experience. Md Med J. 1992;41:315-319.
26. Reid W. Evaluating and treating disabled or impaired colleagues. J Psychiatr Pract. 2007;13:44-48.
From all outward appearances Dr. S, a part-time psychiatrist at an inpatient psychiatric facility and in private practice for 12 years, is living the “perfect life,” with a wife, children, and successful practice.
In retrospect, his drug addiction began insidiously. In college, Dr. S continued to use oxycodone/acetaminophen prescribed for a shoulder injury long after his pain had resolved. He began to use cocaine in residency to help him “get through” the 36-hour call days, and it “helped” him earn the chief resident position because of his heightened energy and concentration. Dr. S’ primary care physician initially prescribed him benzodiazepines for anxiety and to help him sleep. Opiates were prescribed for a musculoskeletal injury. Dr. S obtained prescriptions for these medications from multiple providers. This ultimately escalated to self-prescription using aliases. Dr. S also began to drink heavily each evening.
Dr. S disregards colleagues’ comment about his obvious mood swings, which he attributes to his stressful job and “nagging” wife, despite having a family history of bipolar disorder. He becomes enraged when his wife or friends suggest he seek help. His colleagues whisper behind his back, but for years, no one confronts him about his unpredictable and frequently inappropriate behavior. Eventually, a nurse files a sexual harassment suit against Dr. S, and a patient complains to the medical board that Dr. S exhibited sexually inappropriate behavior during a therapy session.
As physicians, recognizing impairment in our colleagues or ourselves can be difficult. The American Medical Association defines an impaired physician as one who is unable to fulfill personal or professional responsibilities because of psychiatric illness, alcoholism, or drug dependence.1 Impairment is present when a physician is unable to perform in a manner that conforms to acceptable standards of practice, exhibits serious flaws in judgment, and provides incompetent care.1-3
Recognizing when a physician is impaired, deciding whether to report him or her to the state medical board, and referring a colleague for treatment can be challenging. This article will:
- review substance abuse, cognitive decline, and other causes of impairment
- address legal and ethical issues involved in reporting a colleague to the state medical board
- provide resources for physician treatment and assistance.
Physicians and addiction
Chemical dependence is the most frequent disabling illness among physicians,4 and substance abuse is the most common form of impairment that results in discipline by a state medical board.5 An estimated 6% to 8% of physicians abuse drugs, and approximately 14% develop alcohol use disorders; these rates are comparable to those of the general population.5 Psychiatrists, emergency room physicians, and solo practitioners are 3 times more likely to abuse substances than other doctors.6 An obsessive-compulsive personality and other factors may predispose physicians to substance abuse (Table 1).7,8
Alcohol is the most commonly abused substance, followed by opiates, cocaine, and other stimulants.9 Physicians are estimated to use opiates and benzodiazepines at a rate 5 times greater than that of the general public.10,11
An often-hidden problem. Physicians frequently deny substance abuse and many are able to conceal the problem from coworkers, even as their personal lives disintegrate.1,12 Marital and relationship problems may be the first indication of impairment, which gradually spreads to other aspects of their lives (Table 2).1,2,5 A doctor’s professional performance often is the last area to be affected.1,12
Substance abuse in physicians may long go unreported. The clinician’s family may want to protect the physician’s reputation, career, and income. Colleagues may be intimidated, uncertain of their concerns, or fearful for their jobs if they report the physician’s impairment. Patients may be reluctant to report their concerns because they depend on their provider for health care, respect the physician, or deny that a doctor could have a drug or alcohol problem.5
Table 1
Physicians and substance abuse: Predisposing factors
| Obsessive-compulsive personality style |
| Family history of substance use disorders or mental illness |
| Childhood family problems |
| Personal mental illness |
| Sensation-seeking behavior |
| Denial of personal and social problems |
| Perfectionism |
| Idealism |
| Source: References 7,8 |
Table 2
Signs of substance abuse
| Frequent tardiness and absences |
| Unexplained disappearances during working hours |
| Inappropriate behavior |
| Affective lability or irritability |
| Interpersonal conflict |
| Avoidance of peers or supervisors |
| Keeping odd hours |
| Disorganization and forgetfulness |
| Diminished chart completion and work performance |
| Heavy drinking at social functions |
| Unexplained changes in weight or energy level |
| Diminished personal hygiene |
| Slurred or rapid speech |
| Frequently dilated pupils or red and watery eyes and a runny nose |
| Defensiveness, anxiety, apathy, or manipulative behavior |
| Withdrawal from long-standing relationships |
| Source: References 1,2,5 |
Screening for cognitive decline
Many people with cognitive impairment lack insight into their problem and may minimize or deny the degree of their impairment.13 The prevalence of dementia in individuals age ≥65 is 3% to 11%,14 and 18% of physicians are in this age group.15
Ethical, legal, and practical issues arise in determining who, when, and how to screen physicians for cognitive problems. Standard screening exams may not be adequately sensitive for a well-educated physician, and neuropsychological testing may be necessary to detect mild cognitive impairment.13 In addition, the cognitive, visual-spacial, reactivity, reasoning, and calculation skills required for capable medical practice vary among specialties.16
One screening option is a “360-degree review” of information obtained from peers, patients, and non-physician colleagues that the College of Physicians and Surgeons in Alberta, Canada, has incorporated into its Physician’s Achievement Review (PAR) for physicians age ≥65.17 Compiled in a confidential manner and shared with the physician, the 360-degree survey assesses his or her:
- skill and knowledge
- psychosocial functioning
- management skills
- performance
- collegiality.
In Alberta, physicians who score in the lower one-third of the survey are assessed with an on-site evaluation by physicians from his or her specialty appointed by the PAR Director of Practice Improvement.
Alternately, physicians age ≥65 could be required to undergo annual or biannual neuropsychological testing to screen for mild cognitive impairment or other evidence of cognitive decline. In the United States, any screening requirement must be structured to comply with the Age Discrimination in Employment Act.18
If a physician shows evidence of cognitive impairment, the state medical board should initiate closer scrutiny and modify or revoke privileges if indicated. Remediation programs designed to assist impaired physicians may not be effective for those with cognitive impairment because the decline in cognitive functioning associated with illnesses such as Alzheimer’s disease often is progressive.13
‘Disruptive’ physicians
Mental illnesses such as personality or affective disorders, interpersonal problems within or outside the workplace, or other stressors could lead a physician to disrupt the workplace or patient care. Numerous programs have been established across the United States to help evaluate and treat disruptive physicians. Remediation programs can help identify and offer education for “dyscompetent” physicians (see Related Resources).13
To report, or not to report
In a national survey of physicians, only 45% of respondents indicated that they had notified the state licensing board of a colleague they felt was impaired or incompetent, yet almost all (96%) indicated that these individuals should be reported.19 Any duty to report requires, at minimum, that the physician be affected by an illness that impairs his or her cognition, concentration, rapid decision making, and ability to handle emergencies or perform work functions safely.20
Shouten20 cautions that someone who is considering filing a report because of fear of liability if they don’t should balance this concern against potential liability for breaching confidentiality. If there is evidence of an imminent risk or serious harm to the physician or patients, you may be legally required to breach confidentiality. Some states require licensed health practitioners to report acts of professional misconduct, unless the information is obtained solely from directly treating the physician. These requirements apply only within the state, and only to that state’s licensees.20-22
An ethical requirement to report also must be balanced against the obligation to maintain confidentiality. Ethics are largely a matter of individual standards, and individuals’ perceived ethical duties vary.
If you are considering reporting an impaired colleague, learn the laws in your state. If the physician is from another jurisdiction, the law provides little definitive guidance. Shouten recommends focusing on clinical outcomes for the doctor and his or her patients rather than on legal liability.20
Physician health programs
Nationwide directories of physician health programs are available from the Federation of State Physician Health Programs and the Federation of State Medical Boards (see Related Resources). Some programs are affiliated with state licensing boards, some are branches of state medical societies, and some are independent. These programs provide confidential treatment and assistance to practitioners experiencing substance or alcohol abuse, mental illness, or disruptive behavior. Some institutions may offer physicians an employee assistance program.
State medical societies may provide information about accessing a physician health program. Programs sponsored by medical societies almost always are independent of state licensing boards. This arrangement allows physicians to seek help without fear of punishment or censure or revocation of their license. Noncompliance with a physician health program, however, likely will result in being reported to the medical board.
Physician health programs typically employ a rehabilitative approach. Punitive measures such as reporting physicians to the medical board usually are not pursued unless the individual does not comply with treatment and monitoring guidelines. A physician who abuses substances, for example, may be required to complete a residential treatment program, attend support group meetings such as 12-step programs, participate in individual therapy, and undergo random screening for alcohol and illicit drug use.5
Abstinence is the goal of treating clinicians who abuse substances. Physicians have better outcomes than the general population, with reported abstinence rates of 70% to 90% for those who complete treatment;23,24 75% to 85% of physicians who complete rehabilitation and comply with close monitoring and follow-up care are able to return to work.24,25 Acceptance of recovery as a lifelong process, monitoring, and self-vigilance often are necessary to achieve and maintain abstinence.5
Risk factors for relapse include:
- denial of illness
- poor stress-coping and relationship skills
- social and professional isolation
- inability to accept feedback
- complacency and overconfidence
- failure to attend support group meetings
- dysfunctional family dynamics
- feelings of self-pity, blame, and guilt.5
Treating an impaired colleague. Reid26 recommends that psychiatrists should not evaluate or treat a self-referred, potentially impaired physician unless the relationship is strictly clinical. A physician may withhold symptoms, behaviors, or problems because his or her license, malpractice case, or career are at stake.
Advise a physician who requests evaluation or treatment related to license concerns or any legal matter to seek legal counsel. Working with such physician/patients only upon referral by a lawyer, licensing board, or physicians’ health committee provides treating psychiatrists with a clear professional role, allowing them to focus solely on the physician/patient’s treatment needs.
CASE CONTINUED: Extensive help, then success
The medical director of the hospital where Dr. S works refers him to his state’s impaired physician program. After investigating the complaints by the nurse and patient, the medical board suspends Dr. S’ license and requires him to enter a substance abuse treatment program. He completes an intensive residential program for impaired physicians and achieves sobriety from drugs and alcohol. His mood disorder is successfully treated with medications and psychotherapy. The medical board requires Dr. S to have a chaperone present for all visits with patients and submit random urine drug screens once his license is provisionally restored. The medical board also requires Dr. S to undergo ongoing psychiatric care and medication monitoring. He remains abstinent from alcohol and drugs, complies with the medical board’s requirements, and enjoys a productive practice and improved relationship with his family.
Disruptive physicians
- Samenow C, Swiggart W, Spickard A Jr. Consequence of physician disruptive behavior. Tenn Med. 2007;100(11):38-40.
- Leape LL, Fromson JA. Problem doctors: is there a system-level solution? Ann Intern Med. 2006;144(2):107-115.
- Linney BJ. Confronting the disruptive physician. Physician Exec. 1997;23:55-59.
Physician evaluation
- Anfang SA, Faulkner LR, Fromson JA, et al. The American Psychiatric Association’s resource document on guidelines for psychiatric fitness-for-duty evaluations of physicians. J Am Acad Psychiatry Law. 2005;33:85-88.
- Harmon L, Pomm R. Evaluation, treatment, and monitoring of disruptive physicians’ behavior. Psychiatr Ann. 2004; 34:770-774.
Other resources
- Federation of State Physician Health Programs. www.fsphp.org.
- Federation of State Medical Boards: Directory of Physician Assessment and Remedial Education Programs. www.fsmb.org/pdf/RemEdProg.pdf.
- The Center for Professional Health, Vanderbilt University Medical Center. www.mc.vanderbilt.edu/cph.
- Vanderbilt Comprehensive Assessment Program. www.mc.vanderbilt.edu/root/vcap.
- Alcoholics Anonymous. www.aa.org.
- Narcotics Anonymous. www.na.org.
Drug Brand Name
- Oxycodone/acetaminophen • Percocet
Disclosure
The authors report no financial relationship with the manufacturer of any product mentioned in this article or with manufacturers of competing products.
From all outward appearances Dr. S, a part-time psychiatrist at an inpatient psychiatric facility and in private practice for 12 years, is living the “perfect life,” with a wife, children, and successful practice.
In retrospect, his drug addiction began insidiously. In college, Dr. S continued to use oxycodone/acetaminophen prescribed for a shoulder injury long after his pain had resolved. He began to use cocaine in residency to help him “get through” the 36-hour call days, and it “helped” him earn the chief resident position because of his heightened energy and concentration. Dr. S’ primary care physician initially prescribed him benzodiazepines for anxiety and to help him sleep. Opiates were prescribed for a musculoskeletal injury. Dr. S obtained prescriptions for these medications from multiple providers. This ultimately escalated to self-prescription using aliases. Dr. S also began to drink heavily each evening.
Dr. S disregards colleagues’ comment about his obvious mood swings, which he attributes to his stressful job and “nagging” wife, despite having a family history of bipolar disorder. He becomes enraged when his wife or friends suggest he seek help. His colleagues whisper behind his back, but for years, no one confronts him about his unpredictable and frequently inappropriate behavior. Eventually, a nurse files a sexual harassment suit against Dr. S, and a patient complains to the medical board that Dr. S exhibited sexually inappropriate behavior during a therapy session.
As physicians, recognizing impairment in our colleagues or ourselves can be difficult. The American Medical Association defines an impaired physician as one who is unable to fulfill personal or professional responsibilities because of psychiatric illness, alcoholism, or drug dependence.1 Impairment is present when a physician is unable to perform in a manner that conforms to acceptable standards of practice, exhibits serious flaws in judgment, and provides incompetent care.1-3
Recognizing when a physician is impaired, deciding whether to report him or her to the state medical board, and referring a colleague for treatment can be challenging. This article will:
- review substance abuse, cognitive decline, and other causes of impairment
- address legal and ethical issues involved in reporting a colleague to the state medical board
- provide resources for physician treatment and assistance.
Physicians and addiction
Chemical dependence is the most frequent disabling illness among physicians,4 and substance abuse is the most common form of impairment that results in discipline by a state medical board.5 An estimated 6% to 8% of physicians abuse drugs, and approximately 14% develop alcohol use disorders; these rates are comparable to those of the general population.5 Psychiatrists, emergency room physicians, and solo practitioners are 3 times more likely to abuse substances than other doctors.6 An obsessive-compulsive personality and other factors may predispose physicians to substance abuse (Table 1).7,8
Alcohol is the most commonly abused substance, followed by opiates, cocaine, and other stimulants.9 Physicians are estimated to use opiates and benzodiazepines at a rate 5 times greater than that of the general public.10,11
An often-hidden problem. Physicians frequently deny substance abuse and many are able to conceal the problem from coworkers, even as their personal lives disintegrate.1,12 Marital and relationship problems may be the first indication of impairment, which gradually spreads to other aspects of their lives (Table 2).1,2,5 A doctor’s professional performance often is the last area to be affected.1,12
Substance abuse in physicians may long go unreported. The clinician’s family may want to protect the physician’s reputation, career, and income. Colleagues may be intimidated, uncertain of their concerns, or fearful for their jobs if they report the physician’s impairment. Patients may be reluctant to report their concerns because they depend on their provider for health care, respect the physician, or deny that a doctor could have a drug or alcohol problem.5
Table 1
Physicians and substance abuse: Predisposing factors
| Obsessive-compulsive personality style |
| Family history of substance use disorders or mental illness |
| Childhood family problems |
| Personal mental illness |
| Sensation-seeking behavior |
| Denial of personal and social problems |
| Perfectionism |
| Idealism |
| Source: References 7,8 |
Table 2
Signs of substance abuse
| Frequent tardiness and absences |
| Unexplained disappearances during working hours |
| Inappropriate behavior |
| Affective lability or irritability |
| Interpersonal conflict |
| Avoidance of peers or supervisors |
| Keeping odd hours |
| Disorganization and forgetfulness |
| Diminished chart completion and work performance |
| Heavy drinking at social functions |
| Unexplained changes in weight or energy level |
| Diminished personal hygiene |
| Slurred or rapid speech |
| Frequently dilated pupils or red and watery eyes and a runny nose |
| Defensiveness, anxiety, apathy, or manipulative behavior |
| Withdrawal from long-standing relationships |
| Source: References 1,2,5 |
Screening for cognitive decline
Many people with cognitive impairment lack insight into their problem and may minimize or deny the degree of their impairment.13 The prevalence of dementia in individuals age ≥65 is 3% to 11%,14 and 18% of physicians are in this age group.15
Ethical, legal, and practical issues arise in determining who, when, and how to screen physicians for cognitive problems. Standard screening exams may not be adequately sensitive for a well-educated physician, and neuropsychological testing may be necessary to detect mild cognitive impairment.13 In addition, the cognitive, visual-spacial, reactivity, reasoning, and calculation skills required for capable medical practice vary among specialties.16
One screening option is a “360-degree review” of information obtained from peers, patients, and non-physician colleagues that the College of Physicians and Surgeons in Alberta, Canada, has incorporated into its Physician’s Achievement Review (PAR) for physicians age ≥65.17 Compiled in a confidential manner and shared with the physician, the 360-degree survey assesses his or her:
- skill and knowledge
- psychosocial functioning
- management skills
- performance
- collegiality.
In Alberta, physicians who score in the lower one-third of the survey are assessed with an on-site evaluation by physicians from his or her specialty appointed by the PAR Director of Practice Improvement.
Alternately, physicians age ≥65 could be required to undergo annual or biannual neuropsychological testing to screen for mild cognitive impairment or other evidence of cognitive decline. In the United States, any screening requirement must be structured to comply with the Age Discrimination in Employment Act.18
If a physician shows evidence of cognitive impairment, the state medical board should initiate closer scrutiny and modify or revoke privileges if indicated. Remediation programs designed to assist impaired physicians may not be effective for those with cognitive impairment because the decline in cognitive functioning associated with illnesses such as Alzheimer’s disease often is progressive.13
‘Disruptive’ physicians
Mental illnesses such as personality or affective disorders, interpersonal problems within or outside the workplace, or other stressors could lead a physician to disrupt the workplace or patient care. Numerous programs have been established across the United States to help evaluate and treat disruptive physicians. Remediation programs can help identify and offer education for “dyscompetent” physicians (see Related Resources).13
To report, or not to report
In a national survey of physicians, only 45% of respondents indicated that they had notified the state licensing board of a colleague they felt was impaired or incompetent, yet almost all (96%) indicated that these individuals should be reported.19 Any duty to report requires, at minimum, that the physician be affected by an illness that impairs his or her cognition, concentration, rapid decision making, and ability to handle emergencies or perform work functions safely.20
Shouten20 cautions that someone who is considering filing a report because of fear of liability if they don’t should balance this concern against potential liability for breaching confidentiality. If there is evidence of an imminent risk or serious harm to the physician or patients, you may be legally required to breach confidentiality. Some states require licensed health practitioners to report acts of professional misconduct, unless the information is obtained solely from directly treating the physician. These requirements apply only within the state, and only to that state’s licensees.20-22
An ethical requirement to report also must be balanced against the obligation to maintain confidentiality. Ethics are largely a matter of individual standards, and individuals’ perceived ethical duties vary.
If you are considering reporting an impaired colleague, learn the laws in your state. If the physician is from another jurisdiction, the law provides little definitive guidance. Shouten recommends focusing on clinical outcomes for the doctor and his or her patients rather than on legal liability.20
Physician health programs
Nationwide directories of physician health programs are available from the Federation of State Physician Health Programs and the Federation of State Medical Boards (see Related Resources). Some programs are affiliated with state licensing boards, some are branches of state medical societies, and some are independent. These programs provide confidential treatment and assistance to practitioners experiencing substance or alcohol abuse, mental illness, or disruptive behavior. Some institutions may offer physicians an employee assistance program.
State medical societies may provide information about accessing a physician health program. Programs sponsored by medical societies almost always are independent of state licensing boards. This arrangement allows physicians to seek help without fear of punishment or censure or revocation of their license. Noncompliance with a physician health program, however, likely will result in being reported to the medical board.
Physician health programs typically employ a rehabilitative approach. Punitive measures such as reporting physicians to the medical board usually are not pursued unless the individual does not comply with treatment and monitoring guidelines. A physician who abuses substances, for example, may be required to complete a residential treatment program, attend support group meetings such as 12-step programs, participate in individual therapy, and undergo random screening for alcohol and illicit drug use.5
Abstinence is the goal of treating clinicians who abuse substances. Physicians have better outcomes than the general population, with reported abstinence rates of 70% to 90% for those who complete treatment;23,24 75% to 85% of physicians who complete rehabilitation and comply with close monitoring and follow-up care are able to return to work.24,25 Acceptance of recovery as a lifelong process, monitoring, and self-vigilance often are necessary to achieve and maintain abstinence.5
Risk factors for relapse include:
- denial of illness
- poor stress-coping and relationship skills
- social and professional isolation
- inability to accept feedback
- complacency and overconfidence
- failure to attend support group meetings
- dysfunctional family dynamics
- feelings of self-pity, blame, and guilt.5
Treating an impaired colleague. Reid26 recommends that psychiatrists should not evaluate or treat a self-referred, potentially impaired physician unless the relationship is strictly clinical. A physician may withhold symptoms, behaviors, or problems because his or her license, malpractice case, or career are at stake.
Advise a physician who requests evaluation or treatment related to license concerns or any legal matter to seek legal counsel. Working with such physician/patients only upon referral by a lawyer, licensing board, or physicians’ health committee provides treating psychiatrists with a clear professional role, allowing them to focus solely on the physician/patient’s treatment needs.
CASE CONTINUED: Extensive help, then success
The medical director of the hospital where Dr. S works refers him to his state’s impaired physician program. After investigating the complaints by the nurse and patient, the medical board suspends Dr. S’ license and requires him to enter a substance abuse treatment program. He completes an intensive residential program for impaired physicians and achieves sobriety from drugs and alcohol. His mood disorder is successfully treated with medications and psychotherapy. The medical board requires Dr. S to have a chaperone present for all visits with patients and submit random urine drug screens once his license is provisionally restored. The medical board also requires Dr. S to undergo ongoing psychiatric care and medication monitoring. He remains abstinent from alcohol and drugs, complies with the medical board’s requirements, and enjoys a productive practice and improved relationship with his family.
Disruptive physicians
- Samenow C, Swiggart W, Spickard A Jr. Consequence of physician disruptive behavior. Tenn Med. 2007;100(11):38-40.
- Leape LL, Fromson JA. Problem doctors: is there a system-level solution? Ann Intern Med. 2006;144(2):107-115.
- Linney BJ. Confronting the disruptive physician. Physician Exec. 1997;23:55-59.
Physician evaluation
- Anfang SA, Faulkner LR, Fromson JA, et al. The American Psychiatric Association’s resource document on guidelines for psychiatric fitness-for-duty evaluations of physicians. J Am Acad Psychiatry Law. 2005;33:85-88.
- Harmon L, Pomm R. Evaluation, treatment, and monitoring of disruptive physicians’ behavior. Psychiatr Ann. 2004; 34:770-774.
Other resources
- Federation of State Physician Health Programs. www.fsphp.org.
- Federation of State Medical Boards: Directory of Physician Assessment and Remedial Education Programs. www.fsmb.org/pdf/RemEdProg.pdf.
- The Center for Professional Health, Vanderbilt University Medical Center. www.mc.vanderbilt.edu/cph.
- Vanderbilt Comprehensive Assessment Program. www.mc.vanderbilt.edu/root/vcap.
- Alcoholics Anonymous. www.aa.org.
- Narcotics Anonymous. www.na.org.
Drug Brand Name
- Oxycodone/acetaminophen • Percocet
Disclosure
The authors report no financial relationship with the manufacturer of any product mentioned in this article or with manufacturers of competing products.
1. Breiner SJ. The impaired physician. J Med Educ. 1979;54:673.-
2. Talbott GD, Gallegos KV, Angres DH. Impairment and recovery in physicians and other health professionals. In: Graham AW, Schultz TK, eds. Principles of addiction medicine. 2nd ed. Chevy Chase, MD: American Society of Addiction Medicine, Inc; 1998:1263-1277.
3. American Medical Association Council on Mental Health. The sick physician: impairment by psychiatric disorders, including alcoholism and drug dependence. JAMA. 1973;223:684-687.
4. Talbott G, Wright C. Chemical dependency in healthcare professionals. Occup Med. 1987;2:581-591.
5. Baldisseri MR. Impaired healthcare professional. Crit Care Med. 2007;35:S106-116.
6. Mansky PA. Physician health programs and the potentially impaired physician with a substance use disorder. Psychiatr Serv. 1996;47:465-467.
7. Boisaubin EV, Levine RE. Identifying and assisting the impaired physician. Am J Med Sci. 2001;322:31-36.
8. Bissel L, Jones RW. The alcoholic physician: a survey. Am J Psychiatry. 1976;133:1142-1146.
9. Robins L, Reiger D. Psychiatric disorders in America: the Epidemiologic Catchment Area Study. New York, NY: The Free Press; 1991.
10. Gallegos KV, Browne CH, Veit FW, et al. Addiction in anesthesiologists: drug access and patterns of substance abuse. QRB Qual Rev Bull. 1988;14:116-122.
11. Hughes PH, Brandenburg N, Baldwin DC, et al. Prevalence of substance abuse among U.S. physicians. JAMA. 1992;267:2333-2339.
12. Vaillant GE, Clark W, Cyrus C, et al. Prospective study of alcoholism treatment. Eight year follow-up. Am J Med. 1983;75:455-463.
13. LoboPrabhu SM, Molinari VA, Hamilton JD, et al. The aging physician with cognitive impairment: approaches to oversight, prevention and remediation. Am J Geriatr Psychiatry. 2009;17:445-454.
14. U.S. General Accounting Office. Alzheimer’s disease: estimates of prevalence in the United States. Washington, DC: U.S. General Accounting Office; 1998:98. Publication GAO/HEHS-98-16.
15. American Medical Association. Physician characteristics and distribution in the U.S., 2006. Washington, DC: American Medical Association; 2006.
16. Blasier R. The problem of the aging surgeon. Clin Orthop Relat Res. 2009;467:402-411.
17. College of Physicians and Surgeons of Alberta. Physician Achievement Review (PAR) Program. Available at: http://www.cpsa.ab.ca/Services/PARprogram/Overview.aspx. Accessed March 27, 2010.
18. The Age Discrimination in Employment Act. (Vol. Pub. L. No. 90-202, 81 Stat. 602 (Dec. 15, 1967), codified as Chapter 14 of Title 29 of the United States Code, 29 U.S.C. § 621 through 29 U.S.C. § 63), 1967.
19. Campbell EG, Regan S, Gruen RL, et al. Professionalism in medicine: results of a national survey of physicians. Ann Int Med. 2007;147:795-802.
20. Shouten R. Impaired physicians: is there a duty to report to state licensing boards? Harvard Rev Psychiatry. 2000;8:36-39.
21. Mass Gen Laws ch 112 § 5F.
22. NY PHL § 230 (11) (e).
23. Femino J, Nirenberg TD. Treatment outcome studies on physician impairment: a review of the literature. R I Med. 1994;77:345-350.
24. Alpern F, Correnti CE, Dolan TE, et al. A survey of recovering Maryland physicians. Md Med J. 1992;41:301-303.
25. Gallegos KV, Lubin BH, Bowers C, et al. Relapse and recovery: five to ten year follow-up study of chemically dependent physicians—the Georgia experience. Md Med J. 1992;41:315-319.
26. Reid W. Evaluating and treating disabled or impaired colleagues. J Psychiatr Pract. 2007;13:44-48.
1. Breiner SJ. The impaired physician. J Med Educ. 1979;54:673.-
2. Talbott GD, Gallegos KV, Angres DH. Impairment and recovery in physicians and other health professionals. In: Graham AW, Schultz TK, eds. Principles of addiction medicine. 2nd ed. Chevy Chase, MD: American Society of Addiction Medicine, Inc; 1998:1263-1277.
3. American Medical Association Council on Mental Health. The sick physician: impairment by psychiatric disorders, including alcoholism and drug dependence. JAMA. 1973;223:684-687.
4. Talbott G, Wright C. Chemical dependency in healthcare professionals. Occup Med. 1987;2:581-591.
5. Baldisseri MR. Impaired healthcare professional. Crit Care Med. 2007;35:S106-116.
6. Mansky PA. Physician health programs and the potentially impaired physician with a substance use disorder. Psychiatr Serv. 1996;47:465-467.
7. Boisaubin EV, Levine RE. Identifying and assisting the impaired physician. Am J Med Sci. 2001;322:31-36.
8. Bissel L, Jones RW. The alcoholic physician: a survey. Am J Psychiatry. 1976;133:1142-1146.
9. Robins L, Reiger D. Psychiatric disorders in America: the Epidemiologic Catchment Area Study. New York, NY: The Free Press; 1991.
10. Gallegos KV, Browne CH, Veit FW, et al. Addiction in anesthesiologists: drug access and patterns of substance abuse. QRB Qual Rev Bull. 1988;14:116-122.
11. Hughes PH, Brandenburg N, Baldwin DC, et al. Prevalence of substance abuse among U.S. physicians. JAMA. 1992;267:2333-2339.
12. Vaillant GE, Clark W, Cyrus C, et al. Prospective study of alcoholism treatment. Eight year follow-up. Am J Med. 1983;75:455-463.
13. LoboPrabhu SM, Molinari VA, Hamilton JD, et al. The aging physician with cognitive impairment: approaches to oversight, prevention and remediation. Am J Geriatr Psychiatry. 2009;17:445-454.
14. U.S. General Accounting Office. Alzheimer’s disease: estimates of prevalence in the United States. Washington, DC: U.S. General Accounting Office; 1998:98. Publication GAO/HEHS-98-16.
15. American Medical Association. Physician characteristics and distribution in the U.S., 2006. Washington, DC: American Medical Association; 2006.
16. Blasier R. The problem of the aging surgeon. Clin Orthop Relat Res. 2009;467:402-411.
17. College of Physicians and Surgeons of Alberta. Physician Achievement Review (PAR) Program. Available at: http://www.cpsa.ab.ca/Services/PARprogram/Overview.aspx. Accessed March 27, 2010.
18. The Age Discrimination in Employment Act. (Vol. Pub. L. No. 90-202, 81 Stat. 602 (Dec. 15, 1967), codified as Chapter 14 of Title 29 of the United States Code, 29 U.S.C. § 621 through 29 U.S.C. § 63), 1967.
19. Campbell EG, Regan S, Gruen RL, et al. Professionalism in medicine: results of a national survey of physicians. Ann Int Med. 2007;147:795-802.
20. Shouten R. Impaired physicians: is there a duty to report to state licensing boards? Harvard Rev Psychiatry. 2000;8:36-39.
21. Mass Gen Laws ch 112 § 5F.
22. NY PHL § 230 (11) (e).
23. Femino J, Nirenberg TD. Treatment outcome studies on physician impairment: a review of the literature. R I Med. 1994;77:345-350.
24. Alpern F, Correnti CE, Dolan TE, et al. A survey of recovering Maryland physicians. Md Med J. 1992;41:301-303.
25. Gallegos KV, Lubin BH, Bowers C, et al. Relapse and recovery: five to ten year follow-up study of chemically dependent physicians—the Georgia experience. Md Med J. 1992;41:315-319.
26. Reid W. Evaluating and treating disabled or impaired colleagues. J Psychiatr Pract. 2007;13:44-48.